G
Guido Kroemer
Researcher at Institut Gustave Roussy
Publications - 1546
Citations - 294816
Guido Kroemer is an academic researcher from Institut Gustave Roussy. The author has contributed to research in topics: Programmed cell death & Apoptosis. The author has an hindex of 236, co-authored 1404 publications receiving 246571 citations. Previous affiliations of Guido Kroemer include Karolinska Institutet & Spanish National Research Council.
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Journal ArticleDOI
Molecular and Translational Classifications of DAMPs in Immunogenic Cell Death
Abhishek D. Garg,Lorenzo Galluzzi,Lionel Apetoh,Lionel Apetoh,Thaïs Baert,Raymond B. Birge,José Manuel Bravo-San Pedro,Karine Breckpot,David Brough,Ricardo A. Chaurio,Mara Cirone,An Coosemans,Pierre Coulie,Dirk De Ruysscher,Luciana Dini,Peter de Witte,Aleksandra M. Dudek-Peric,Alberto Faggioni,Jitka Fucikova,Udo S. Gaipl,Jakub Golab,Marie-Lise Gougeon,Michael R. Hamblin,Akseli Hemminki,Martin Herrmann,James W. Hodge,Oliver Kepp,Guido Kroemer,Dmitri V. Krysko,Walter Land,Frank Madeo,Angelo A. Manfredi,Stephen R. Mattarollo,Christian Maueröder,Nicolò Merendino,Gabriele Multhoff,Thomas Pabst,Jean-Ehrland Ricci,Chiara Riganti,Erminia Romano,Nicole Rufo,Mark J. Smyth,Mark J. Smyth,Jürgen Sonnemann,Radek Spisek,John Stagg,Erika Vacchelli,Peter Vandenabeele,Lien Vandenberk,Benoît Van den Eynde,Stefaan Van Gool,Francesca Velotti,Laurence Zitvogel,Patrizia Agostinis +53 more
TL;DR: The main molecular, immunological, preclinical, and clinical aspects of ICD are summarized and tabulate in an attempt to capture the essence of this phenomenon, and identify future challenges for this rapidly expanding field of investigation.
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Autophagy-Dependent Ferroptosis: Machinery and Regulation
TL;DR: In this article, the role of autophagy, especially selective types, in driving cells toward ferroptotic death was explored and the functional interactions between metabolism, immunity, and cell death were explored.
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Contribution of IL-17-producing gamma delta T cells to the efficacy of anticancer chemotherapy.
Yuting Ma,Laetitia Aymeric,Clara Locher,Stephen R. Mattarollo,Nicolas F. Delahaye,Pablo Pereira,Laurent Boucontet,Lionel Apetoh,François Ghiringhelli,Noelia Casares,Juan José Lasarte,Goro Matsuzaki,Koichi Ikuta,Bernard Ryffel,Kamel Benlagha,Antoine Tesniere,Nicolas Ibrahim,Julie Déchanet-Merville,Nathalie Chaput,Mark J. Smyth,Guido Kroemer,Laurence Zitvogel +21 more
TL;DR: IL-17 production by γδ T cells is required for tumor cell infiltration by IFN-γ–producing CD8+ T cells and inhibition of tumor growth in response to anthracyclines.
Journal ArticleDOI
Alternatively spliced NKp30 isoforms affect the prognosis of gastrointestinal stromal tumors
Nicolas F. Delahaye,Sylvie Rusakiewicz,Sylvie Rusakiewicz,Isabelle Martins,Cédric Ménard,Cédric Ménard,Stephan Roux,Stephan Roux,Luc Lyonnet,Pascale Paul,Matthieu Sarabi,Matthieu Sarabi,Nathalie Chaput,Nathalie Chaput,Michaela Semeraro,Michaela Semeraro,Véronique Minard-Colin,Véronique Minard-Colin,Vichnou Poirier-Colame,Kariman Chaba,Kariman Chaba,Caroline Flament,Caroline Flament,Véronique Baud,Véronique Baud,Véronique Baud,Hélène Authier,Hélène Authier,Hélène Authier,Saadia Kerdine-Römer,Marc Pallardy,Isabelle Cremer,Isabelle Cremer,Laetitia Peaudecerf,Benedita Rocha,Dominique Valteau-Couanet,Dominique Valteau-Couanet,Javier Celis Gutierrez,Jacques A. Nunès,Frédéric Commo,Frédéric Commo,Sylvie Bonvalot,Nicolas Ibrahim,Philippe Terrier,Paule Opolon,Paule Opolon,Cristina Bottino,Cristina Bottino,Alessandro Moretta,Jan Tavernier,Pascal Rihet,Jean Michel Coindre,Jean-Yves Blay,Nicolas Isambert,Jean Franãois Emile,Eric Vivier,A. Lecesne,Guido Kroemer,Laurence Zitvogel +58 more
TL;DR: The genetically determined NKp30 status predicts the clinical outcomes of individuals with GIST independently from KIT mutation, and predominant expression of the immunosuppressiveNKp30c isoform was associated with reduced survival of subjects.
Journal ArticleDOI
Mitotic catastrophe constitutes a special case of apoptosis whose suppression entails aneuploidy
Maria Castedo,Jean-Luc Perfettini,Thomas Roumier,Alexander Valent,Hana Raslova,Kenichi Yakushijin,David A. Horne,Jean Feunteun,Gilbert M. Lenoir,René H. Medema,William Vainchenker,Guido Kroemer +11 more
TL;DR: A molecular pathway through which DNA damage, failure to arrest the cell cycle and inhibition of apoptosis can favor the occurrence of cytogenetic abnormalities that are likely to participate in oncogenesis is delineated.