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Günter U. Höglinger

Researcher at Hannover Medical School

Publications -  309
Citations -  19994

Günter U. Höglinger is an academic researcher from Hannover Medical School. The author has contributed to research in topics: Progressive supranuclear palsy & Medicine. The author has an hindex of 54, co-authored 261 publications receiving 16054 citations. Previous affiliations of Günter U. Höglinger include Pierre-and-Marie-Curie University & University of Bern.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Clinical diagnosis of progressive supranuclear palsy: The movement disorder society criteria

Günter U. Höglinger, +47 more
- 01 Jun 2017 - 
TL;DR: Clinical diagnostic criteria, published in 1996 by the National Institute of Neurological Disorders and Stroke/Society for PSP have excellent specificity, but their sensitivity is limited for variant PSP syndromes with presentations other than Richardson's syndrome.
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Dopamine depletion impairs precursor cell proliferation in Parkinson disease

Günter U. Höglinger, +6 more
- 01 Jul 2004 - 
TL;DR: In this article, the authors provided ultrastructural evidence showing that highly proliferative precursors in the adult subependymal zone express dopamine receptors and receive dopaminergic afferents.
Journal ArticleDOI

Identification of common variants influencing risk of the tauopathy progressive supranuclear palsy

Günter U. Höglinger, +140 more
- 01 Jul 2011 - 
TL;DR: Two independent variants in MAPT affecting risk for PSP are confirmed, one of which influences MAPT brain expression and the genes implicated encode proteins for vesicle-membrane fusion at the Golgi-endosomal interface and for a myelin structural component.
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Chronic systemic complex I inhibition induces a hypokinetic multisystem degeneration in rats.

Günter U. Höglinger, +10 more
- 01 Feb 2003 - 
TL;DR: The data suggest that a generalized mitochondrial failure may be implicated in atypical parkinsonian syndromes but do not support the hypothesis that an generalized complex I inhibition results in the rather selective nigral lesion observed in Parkinson's disease.