scispace - formally typeset
H

Haichao Wang

Researcher at The Feinstein Institute for Medical Research

Publications -  264
Citations -  45991

Haichao Wang is an academic researcher from The Feinstein Institute for Medical Research. The author has contributed to research in topics: HMGB1 & Proinflammatory cytokine. The author has an hindex of 81, co-authored 255 publications receiving 40235 citations. Previous affiliations of Haichao Wang include Central South University & New York University.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Vagus nerve stimulation attenuates the systemic inflammatory response to endotoxin

Lyudmila V. Borovikova, +9 more
- 25 May 2000 - 
TL;DR: Direct electrical stimulation of the peripheral vagus nerve in vivo during lethal endotoxaemia in rats inhibited TNF synthesis in liver, attenuated peak serum TNF amounts, and prevented the development of shock.
Journal ArticleDOI

HMG-1 as a Late Mediator of Endotoxin Lethality in Mice

Haichao Wang, +18 more
- 09 Jul 1999 - 
TL;DR: High mobility group-1 (HMG-1) protein was found to be released by cultured macrophages more than 8 hours after stimulation with endotoxin, TNF, or IL-1, and showed increased serum levels after endotoxin exposure, suggesting that this protein warrants investigation as a therapeutic target.
Journal ArticleDOI

Nicotinic acetylcholine receptor alpha7 subunit is an essential regulator of inflammation.

Hong Wang, +12 more
- 23 Jan 2003 - 
TL;DR: It is reported that the nicotinic acetylcholine receptor α7 subunit is essential for inhibiting cytokine synthesis by the cholinergic anti-inflammatory pathway.