Hannan Tahir

TL;DR: Simulation results suggest that the growth of the restenotic lesion is strongly dependent on the stent strut cross-sectional profile, and a strong correlation between strut thickness and the rate of smooth muscle cell proliferation has been observed.

TL;DR: The data indicate a positive correlation between the neointimal growths and strut deployment depths in the presence of a functional endothelium, in qualitative agreement with in-vivo data.

TL;DR: Comparison between in vivo and in silico results suggests that endothelial growth does not occur from the proximal and distal ends alone, but is more consistent with the assumption of a random seeding process.

TL;DR: It is hypothesized that a deeper stent deployment results in on average larger fenestrae in the elastic lamina, allowing easier migration of SMCs into the lumen, and that growth of the neointimal lesions owing to SMC proliferation is strongly dependent on the initial number of migrated cells, which form an initial condition for the later phase of the vascular repair.

TL;DR: In this article, the authors developed an agent-based model and compared the impact of personal protective equipment (PPE), screening of healthcare workers (HCWs), contact tracing of symptomatic HCWs and restricting HCWs from working in multiple units (HCW cohorting) on nosocomial SARS-CoV-2 transmission.