scispace - formally typeset
H

Heidi Declercq

Researcher at Ghent University

Publications -  147
Citations -  4691

Heidi Declercq is an academic researcher from Ghent University. The author has contributed to research in topics: Surface modification & Self-healing hydrogels. The author has an hindex of 30, co-authored 144 publications receiving 3557 citations. Previous affiliations of Heidi Declercq include Katholieke Universiteit Leuven & Ghent University Hospital.

Papers
More filters
Journal ArticleDOI

Nanofat Grafting: Basic Research and Clinical Applications

TL;DR: In clinical situations, nanofat seems to be suitable for skin rejuvenation purposes, and might become a new concept in the lipofilling area.
Journal ArticleDOI

Increased fibroblast proliferation induced by light emitting diode and low power laser irradiation.

TL;DR: LED and LLL irradiation resulted in an increased fibroblast proliferation in vitro, postulates possible stimulatory effects on wound healing in vivo at the applied dosimetric parameters.
Journal ArticleDOI

Isolation, proliferation and differentiation of osteoblastic cells to study cell/biomaterial interactions: comparison of different isolation techniques and source.

TL;DR: It is demonstrated that considering the isolation method, proliferation capacity and the osteogenic potential, the enzymatically released fetal calvaria cells are most satisfactory to study cell/biomaterial interactions.
Journal ArticleDOI

Calcification as an indicator of osteoinductive capacity of biomaterials in osteoblastic cell cultures.

TL;DR: It is demonstrated that dystrophic calcification can not be distinguished from cell-mediated calcification with von Kossa, FTIR and X-ray diffractometry and primary osteoblastic cells capable of forming nodules are recommended to evaluate the osteoinductive properties of biomaterials.
Journal ArticleDOI

The role of endothelial cells in the vasculopathy of systemic sclerosis: A systematic review

TL;DR: This systematic review emphasizes the growing evidence that SSc is primarily a vascular disease where EC dysfunction is present and prominent in different aspects of cell survival, angiogenesis and vasculogenesis and where disturbed interactions between ECs and various other cells contribute to SSc vasculopathy.