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Helen K. W. Law

Researcher at Hong Kong Polytechnic University

Publications -  96
Citations -  10627

Helen K. W. Law is an academic researcher from Hong Kong Polytechnic University. The author has contributed to research in topics: Autophagy & T cell. The author has an hindex of 36, co-authored 93 publications receiving 8603 citations. Previous affiliations of Helen K. W. Law include Li Ka Shing Faculty of Medicine, University of Hong Kong & University of Melbourne.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Chemokine up-regulation in SARS-coronavirus-infected, monocyte-derived human dendritic cells.

Helen K. W. Law, +8 more
- 01 Oct 2005 - 
TL;DR: No increase in virus titer was detected in infected DCs and cell-culture supernatant, confirming that virus replication was incomplete and the lack of antiviral cytokine response against a background of intense chemokine up-regulation could represent a mechanism of immune evasion by SARS-CoV.
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Mannose-Binding Lectin in Severe Acute Respiratory Syndrome Coronavirus Infection

W. K. Eddie Ip, +15 more
- 15 May 2005 - 
TL;DR: It is suggested that MBL contributes to the first-line host defense against SARS-CoV and that M BL deficiency is a susceptibility factor for acquisition of SARS.
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Innate sensing of HIV-infected cells.

Alice Lepelley, +12 more
- 17 Feb 2011 - 
TL;DR: It is shown that infected lymphocytes are more potent inducers of IFN than virions, and that the IRF3 pathway, through a process requiring access of incoming viral material to the cytoplasm, allows sensing of HIV-infected lymphocytes.