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Hiromi Sesaki

Researcher at Johns Hopkins University School of Medicine

Publications -  161
Citations -  19784

Hiromi Sesaki is an academic researcher from Johns Hopkins University School of Medicine. The author has contributed to research in topics: Mitochondrion & Mitochondrial fission. The author has an hindex of 56, co-authored 147 publications receiving 15718 citations. Previous affiliations of Hiromi Sesaki include Johns Hopkins University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Mitochondrial Dynamics Controls T Cell Fate Through Metabolic Programming

Michael D. Buck, +24 more
- 30 Jun 2016 - 
TL;DR: The data suggest that, by altering cristae morphology, fusion in TM cells configures electron transport chain (ETC) complex associations favoring oxidative phosphorylation (OXPHOS) and FAO, while fission in TE cells leads to cristsae expansion, reducing ETC efficiency and promoting aerobic glycolysis.
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The dynamin-related GTPase Drp1 is required for embryonic and brain development in mice

Junko Wakabayashi, +7 more
- 21 Sep 2009 - 
TL;DR: Brain-specific Drp1 knockout mice demonstrate thatDrp1-mediated organelle division is important for development, mitochondrial morphogenesis, and apoptosis.
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Direct Membrane Association Drives Mitochondrial Fission by the Parkinson Disease-associated Protein α-Synuclein

Ken Nakamura, +15 more
- 10 Jun 2011 - 
TL;DR: It is shown that the expression of α-synuclein in mammalian cells, including neurons in vitro and in vivo, causes the fragmentation of mitochondria.