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Ho-Joong Youn

Researcher at Catholic University of Korea

Publications -  155
Citations -  1621

Ho-Joong Youn is an academic researcher from Catholic University of Korea. The author has contributed to research in topics: Coronary artery disease & Medicine. The author has an hindex of 20, co-authored 145 publications receiving 1367 citations. Previous affiliations of Ho-Joong Youn include St Mary's Hospital.

Papers
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Journal ArticleDOI

Peroxisome proliferator-activated receptor-γ activation attenuates cardiac fibrosis in type 2 diabetic rats: the effect of rosiglitazone on myocardial expression of receptor for advanced glycation end products and of connective tissue growth factor

Sang-Hyun Ihm, +6 more
- 01 May 2010 - 
TL;DR: Rosiglitazone treatment significantly improved E/A ratio in serial echocardiography assessment, and reduced LV collagen volume fraction as demonstrated by picrosirius red staining support the potential of PPAR-γ agonists as antifibrotic agents in diabetic cardiomyopathy.
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Metabolically obese status with normal weight is associated with both the prevalence and severity of angiographic coronary artery disease.

Beom-June Kwon, +11 more
- 01 Jul 2013 - 
TL;DR: MONW or MO is associated with both the prevalence and severity of angiographic CAD after adjustment for age and sex and MONW is independently associated with the severity ofAngiographic coronary artery disease irrespective of DM.
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Demonstration of coronary artery flow using transthoracic Doppler echocardiography

Ho-Joong Youn, +1 more
- 01 Feb 2004 - 
TL;DR: Measurement of coronary flow reserve may impact diagnosis or clinical treatment in those: with anginal chest pain and angiographically normal coronary arteries; with intermediate-grade coronary obstruction; and who have had an attempt at revascularization and the effectiveness of the therapy is uncertain.
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Pathophysiology and preventive strategies of anthracycline-induced cardiotoxicity

Woo-Baek Chung, +1 more
- 01 Jul 2016 - 
TL;DR: Prevention is a more effective approach than treatment of cardiotoxicity after symptomatic or asymptomatic cardiac dysfunction develops and strategies for protecting the myocardium from anthracycline are reviewed.
Journal ArticleDOI

Induction of caspase-independent apoptosis in H9c2 cardiomyocytes by adriamycin treatment.

Ho-Joong Youn, +6 more
- 01 Feb 2005 - 
TL;DR: Caspase inhibition effectively blocked the apoptosis induced by H2O2 in H9c2 cells, as determined by an MTT assay or microscopy, and p53 expression was increased by adriamycin, suggesting a role for p53 in adRIamycin-induced caspase-independent apoptosis in cardiac toxicity.