Hyun-Chul Kim

Bio: Hyun-Chul Kim is an academic researcher from Inha University. The author has contributed to research in topics: Large Hadron Collider & Lepton. The author has an hindex of 176, co-authored 4076 publications receiving 183227 citations. Previous affiliations of Hyun-Chul Kim include Konkuk University & University of Edinburgh.

Search for the standard model Higgs boson decaying to bottom quarks in pp collisions at √{ s} = 7 TeV

Abstract: A search for the standard model Higgs boson (H) decaying to a b-quark pair when produced in association with weak vector bosons (V) is reported for the following modes: W(mu,nu)H, W(e,nu)H, Z(mu,mu)H, Z(e,e)H and Z(nu,nu)H. The search is performed in a data sample corresponding to an integrated luminosity of 4.7 inverse femtobarns, recorded by the CMS detector in proton-proton collisions at the LHC with a center-of-mass energy of 7 TeV. No significant excess of events above the expectation from background is observed. Upper limits on the VH production cross section times the H to b-quark pair branching ratio, with respect to the expectations for a standard model Higgs boson, are derived for a Higgs boson in the mass range 110-135 GeV. In this range, the observed 95% confidence level upper limits vary from 3.4 to 7.5 times the standard model prediction; the corresponding expected limits vary from 2.7 to 6.7.

Search for charged higgs boson decays of the top quark using hadronic decays of the tau lepton

Abstract: This Letter describes a direct search for charged Higgs boson production in p{ovr p} collisions at {radical}(s)=1.8 TeV recorded by the Collider Detector at Fermilab. Two-Higgs-double extensions to the standard model predict the existence of charged Higgs bosons (H{sup {plus_minus}}) . In such models, the branching fraction for top quarks scr(B)(t{r_arrow}H{sup +}b{r_arrow}{tau}{sup +} {nu}b) can be large. This search uses the hadronic decays of the tau lepton in this channel to significantly extend previous limits on H{sup {plus_minus}} production. {copyright} {ital 1997} {ital The American Physical Society}

I and i

Abstract: There is, I think, something ethereal about i —the square root of minus one. I remember first hearing about it at school. It seemed an odd beast at that time—an intruder hovering on the edge of reality. Usually familiarity dulls this sense of the bizarre, but in the case of i it was the reverse: over the years the sense of its surreal nature intensified. It seemed that it was impossible to write mathematics that described the real world in …

Fast parallel algorithms for short-range molecular dynamics

Abstract: Three parallel algorithms for classical molecular dynamics are presented. The first assigns each processor a fixed subset of atoms; the second assigns each a fixed subset of inter-atomic forces to compute; the third assigns each a fixed spatial region. The algorithms are suitable for molecular dynamics models which can be difficult to parallelize efficiently—those with short-range forces where the neighbors of each atom change rapidly. They can be implemented on any distributed-memory parallel machine which allows for message-passing of data between independently executing processors. The algorithms are tested on a standard Lennard-Jones benchmark problem for system sizes ranging from 500 to 100,000,000 atoms on several parallel supercomputers--the nCUBE 2, Intel iPSC/860 and Paragon, and Cray T3D. Comparing the results to the fastest reported vectorized Cray Y-MP and C90 algorithm shows that the current generation of parallel machines is competitive with conventional vector supercomputers even for small problems. For large problems, the spatial algorithm achieves parallel efficiencies of 90% and a 1840-node Intel Paragon performs up to 165 faster than a single Cray C9O processor. Trade-offs between the three algorithms and guidelines for adapting them to more complex molecular dynamics simulations are also discussed.

疟原虫var基因转换速率变化导致抗原变异[英]／Paul H, Robert P, Christodoulou Z, et al//Proc Natl Acad Sci U S A

Abstract: 抗原变异可使得多种致病微生物易于逃避宿主免疫应答。表达在感染红细胞表面的恶性疟原虫红细胞表面蛋白1（PfPMP1）与感染红细胞、内皮细胞、树突状细胞以及胎盘的单个或多个受体作用，在黏附及免疫逃避中起关键的作用。每个单倍体基因组var基因家族编码约60种成员，通过启动转录不同的var基因变异体为抗原变异提供了分子基础。