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Isabella Panfoli
Researcher at University of Genoa
Publications - 159
Citations - 2940
Isabella Panfoli is an academic researcher from University of Genoa. The author has contributed to research in topics: ATP synthase & Mitochondrion. The author has an hindex of 28, co-authored 149 publications receiving 2507 citations.
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Journal Article
Antioxidant role of metallothioneins: a comparative overview.
TL;DR: As the relevance of redox processes in cell signaling is becoming more and more evident, a search for MT effects on redox signaling could represent a turning point in the understanding of the functional role of these protein.
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Evidence for aerobic ATP synthesis in isolated myelin vesicles
Silvia Ravera,Isabella Panfoli,Daniela Calzia,Maria Grazia Aluigi,Paolo Bianchini,Alberto Diaspro,Gianluigi Mancardi,Alessandro Morelli +7 more
TL;DR: Evidence is presented that isolated myelin vesicles (IMV) are able to consume O(2) and produce ATP through the operation of a proton gradient across their membranes, which suggests that myelin sheath may be the site of oxygen absorption and aerobic metabolism for the axons.
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Guanylate cyclase in rod outer segments of the toad retina. Effect of light and Ca2
TL;DR: Visual transduction Guanylate cyclase Rhodopsin cyclic GMP metabolism Photoreceptor Visual transduction
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Evidence for aerobic metabolism in retinal rod outer segment disks
Isabella Panfoli,Daniela Calzia,Paolo Bianchini,Silvia Ravera,Alberto Diaspro,Giovanni Candiano,Angela Bachi,Massimiliano Monticone,Maria Grazia Aluigi,Stefano Barabino,Giovanni Calabria,Maurizio Rolando,Carlo Tacchetti,Alessandro Morelli,I.M. Pepe +14 more
TL;DR: Overall, data indicate that an oxidative phosphorylation occurs in rod OS, which do not contain mitochondria, thank to the presence of ectopically located mitochondrial proteins.
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Oxidative Stress as a Primary Risk Factor for Brain Damage in Preterm Newborns.
Isabella Panfoli,Giovanni Candiano,Mariya Malova,Laura Costanza De Angelis,Valentina Cardiello,Giuseppe Buonocore,Luca A. Ramenghi +6 more
TL;DR: The role of free radical oxidative damage to the newborn brain is reviewed and a mechanism of oxidative injury is proposed, taking into consideration the particular maturation-dependent vulnerability of the oligodendrocyte precursors.