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J. William O. Ballard

Researcher at University of New South Wales

Publications -  94
Citations -  7054

J. William O. Ballard is an academic researcher from University of New South Wales. The author has contributed to research in topics: Mitochondrial DNA & Population. The author has an hindex of 32, co-authored 88 publications receiving 6236 citations. Previous affiliations of J. William O. Ballard include University of Melbourne & Commonwealth Scientific and Industrial Research Organisation.

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The incomplete natural history of mitochondria

TL;DR: A critical examination of the neglected biology of mitochondria is carried out and several surprising gaps in the state of the authors' knowledge about this important organelle are pointed out.
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Lifespan and reproduction in Drosophila: New insights from nutritional geometry

TL;DR: The use of recent techniques in nutrition research to quantify the detailed relationship between diet, nutrient intake, lifespan, and reproduction in Drosophila melanogaster indicates a role for both direct costs of reproduction and other deleterious consequences of ingesting high levels of protein.
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The Ratio of Macronutrients, Not Caloric Intake, Dictates Cardiometabolic Health, Aging, and Longevity in Ad Libitum-Fed Mice.

TL;DR: The Geometric Framework, a state-space nutritional modeling method, was used to measure interactive effects of dietary energy, protein, fat, and carbohydrate on food intake, cardiometabolic phenotype, and longevity in mice fed one of 25 diets ad libitum, suggesting that longevity can be extended in ad Libitum-fed animals by manipulating the ratio of macronutrients.
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The Population Biology of Mitochondrial DNA and Its Phylogenetic Implications

TL;DR: It is argued that developing a fuller understanding of the biology of mitochondria is essential for the rigorous application of mtDNA to inferences about the evolutionary history of species or populations.
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Is mitochondrial DNA a strictly neutral marker

TL;DR: Variation and change in mitochondrial DNA (mtDNA) is often assumed to conform to a constant mutation rate equilibrium neutral model of molecular evolution, however, recent evidence indicates that the assumptions underlying this model are frequently violated.