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Jack L. Lewis

Researcher at University of Minnesota

Publications -  183
Citations -  11098

Jack L. Lewis is an academic researcher from University of Minnesota. The author has contributed to research in topics: Anterior cruciate ligament & Ligament. The author has an hindex of 48, co-authored 183 publications receiving 10429 citations. Previous affiliations of Jack L. Lewis include Clemson University & University of Delaware.

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Book ChapterDOI

Dislocations after total hip-replacement arthroplasties.

TL;DR: It was found that anterior dislocations were associated with increased acetabular-component anteversion and there was no significant correlation between cup-orientation angle and posterior dislocation.
Journal ArticleDOI

Noncontact anterior cruciate ligament injuries: risk factors and prevention strategies.

TL;DR: The risk factors for noncontact ACL injuries fall into four distinct categories: environmental, anatomic, hormonal, and biomechanical as discussed by the authors, and early data on existing neuromuscular training programs suggest that enhancing body control may decrease ACL injuries in women.
Journal ArticleDOI

Properties and an Anisotropic Model of Cancellous Bone From the Proximal Tibial Epiphysis

TL;DR: A finite element model is proposed, based on experimental observations, which enables one to predict the elastic constants of cylindrically structured cancellous bone in the tibia from morphological measurements in the transverse plane.
Journal ArticleDOI

Characteristics of synergic relations during isometric contractions of human elbow muscles

TL;DR: The similarity of activation patterns indicates that elbow torque was the principal determining factor in muscle synergies, and is suggested that they are best understood on the basis of a model which encodes limb torque in premotor neurons.
Journal ArticleDOI

The effects of grade III posterolateral knee complex injuries on anterior cruciate ligament graft force. A biomechanical analysis.

TL;DR: This study supports the clinical observation that untreated grade III posterolateral structure injuries contribute to anterior cruciate ligament graft failure by allowing higher forces to stress the graft.