Jacopo M. Legramante

TL;DR: Very intensive endurance training shifted the cardiovascular autonomic modulation from a parasympathetic toward a sympathetic predominance, and this finding should be interpreted within the context of the substantial role played by the sympathetic nervous system in increasing cardiovascular performance at peak training.

TL;DR: Exercise training increases BRS and heart rate variability in patients with coronary artery disease and improved cardiac autonomic function might add to the other benefits of exercise training in secondary prevention of ischemic heart disease.

TL;DR: The bradycardia that occurs during postexercise muscle ischemia despite the maintained sympathetic stimulus may be explained by a baroreflex-mediated increase in parasympathetic outflow to the sinoatrial node that overpowers the metabore flexor-induced cardiac sympathetic activation.

TL;DR: The data suggest that the central command and muscle chemoreflex act to preserve the BRS, possibly "resetting" the baroreceptor-cardiac response relationship, whereas stimulation of mechanosensitive receptors appears capable of modifying the integrated baroreflex control of sinus node function in humans.

TL;DR: Results show that the spontaneous baroreflex method provides good BRS reproducibility under various stimuli that affect the neural control of circulation differently and suggest that BRS variability is dependent neither on haemodynamic modifications nor on the degree of barore Flex engagement, but it seems to reflect an inherent feature of the way in which arterial barore flexes modulate the heart period.