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James S. Babb

Researcher at New York University

Publications -  369
Citations -  28256

James S. Babb is an academic researcher from New York University. The author has contributed to research in topics: Magnetic resonance imaging & Diffusion MRI. The author has an hindex of 84, co-authored 360 publications receiving 24937 citations. Previous affiliations of James S. Babb include University of York & Fox Chase Cancer Center.

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Fractionated but not single dose radiotherapy induces an immune-mediated abscopal effect when combined with anti-CTLA-4 antibody

M. Zahidunnabi Dewan, +6 more
- 01 Sep 2009 - 
TL;DR: Fractionated but not single-dose radiotherapy induces an abscopal effect when in combination with anti–CTLA-4 antibody in two preclinical carcinoma models.
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Ionizing radiation inhibition of distant untreated tumors (abscopal effect) is immune mediated

Sandra Demaria, +6 more
- 01 Mar 2004 - 
TL;DR: The results demonstrate that the abscopal effect is in part immune mediated and that T cells are required to mediate distant tumor inhibition induced by radiation.
Journal Article

Glioma grading: sensitivity, specificity, and predictive values of perfusion MR imaging and proton MR spectroscopic imaging compared with conventional MR imaging

Meng Law, +8 more
- 01 Nov 2003 - 
TL;DR: The rCBV measurements had the most superior diagnostic performance (either with or without metabolite ratios) in predicting glioma grade and can be used in a clinical setting to evaluate tumors preoperatively for histologic grade and provide a means for guiding treatment and predicting postoperative patient outcome.
Journal Article

Immune-mediated inhibition of metastases after treatment with local radiation and CTLA-4 blockade in a mouse model of breast cancer.

Sandra Demaria, +7 more
- 15 Jan 2005 - 
TL;DR: The combination of local RT with CTLA-4 blockade is a promising new immunotherapeutic strategy against poorly immunogenic metastatic cancers.
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Radiation-induced CXCL16 release by breast cancer cells attracts effector T cells.

Satoko Matsumura, +10 more
- 01 Sep 2008 - 
TL;DR: It is demonstrated that IR markedly enhanced the secretion by mouse and human breast cancer cells of CXCL16, a chemokine that binds to CXCR6 on Th1 and activated CD8 effector T cells, and plays an important role in their recruitment to sites of inflammation.