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Janne Goossens

Researcher at Katholieke Universiteit Leuven

Publications -  10
Citations -  166

Janne Goossens is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: Athletes & Medicine. The author has an hindex of 4, co-authored 7 publications receiving 98 citations.

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Serum neurofilament light chain levels as a marker of upper motor neuron degeneration in patients with Amyotrophic Lateral Sclerosis.

TL;DR: The aims of this study were to further explore the prognostic and diagnostic performances of serum N fL to discriminate between patients with ALS and ALS mimics, and to investigate the relationship between serum NfL with motor neuron degeneration.
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Inflammatory markers in cerebrospinal fluid: independent prognostic biomarkers in amyotrophic lateral sclerosis?

TL;DR: Findings show that inflammation in patients with ALS reflects the disease progression as an independent predictor of survival, and encourage the use of inflammatory markers in patient stratification and as surrogate markers of therapy response in clinical trials.
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Serum neurofilament heavy chains as early marker of motor neuron degeneration.

TL;DR: To determine whether serum phosphorylated neurofilament heavy chain (pNfH) levels are elevated before patients were diagnosed with sporadic or familial ALS, and what the prognostic value of these prediagnostic pNFH levels is.
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Air Pollution and the Airways: Lessons from a Century of Human Urbanization

TL;DR: The goal of this review is to give an overview of the recent data on the effects of air pollution on healthy and diseased airways or models of airway disease, such as asthma or chronic obstructive pulmonary disease, both in mice and humans.
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Early-onset airway damage in early-career elite athletes: A risk factor for exercise-induced bronchoconstriction.

TL;DR: Exercise-induced bronchoconstriction is present in 24.5% of 12-13 years old early-career elite athletes and Epithelial damage might be an early phase hallmark in the development of EIB, in the absence of overt cellular inflammation.