J
Jia-Wei Min
Researcher at University of Texas Health Science Center at Houston
Publications - 23
Citations - 691
Jia-Wei Min is an academic researcher from University of Texas Health Science Center at Houston. The author has contributed to research in topics: Neuroprotection & Vitexin. The author has an hindex of 11, co-authored 20 publications receiving 457 citations. Previous affiliations of Jia-Wei Min include University of South Dakota & South Central University for Nationalities.
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Journal ArticleDOI
A review on the pharmacological effects of vitexin and isovitexin.
TL;DR: This review summarized recent findings on various pharmacological activities and associative signalling pathways of vitexin and isoviteXin to provide a reference for future research and clinical applications.
Journal ArticleDOI
Vitexin reduces hypoxia–ischemia neonatal brain injury by the inhibition of HIF-1alpha in a rat pup model
Jia-Wei Min,Jiang-Jian Hu,Miao He,Russell M. Sanchez,Wen-Xian Huang,Yu-Qiang Liu,Najeeb Bassam Bsoul,Song Han,Jun Yin,Wanhong Liu,Xiaohua He,Biwen Peng +11 more
TL;DR: The data indicate early HIF-1α inhibition with vitexin provides both acute and long-term neuroprotection in the developing brain after neonatal HI injury, as seen by decreased infarct volume evaluated at 48 h post-HI.
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TRPV1 mediates astrocyte activation and interleukin-1β release induced by hypoxic ischemia (HI).
Xing-Liang Yang,Xin Wang,Lin Shao,Guang-Tong Jiang,Jia-Wei Min,Xi-Yu Mei,Xiaohua He,Wanhong Liu,Wen-Xian Huang,Biwen Peng +9 more
TL;DR: These findings provide mechanistic insights into TRPV1-mediated brain damage and neurobehavioral disorders caused by neonatal HI and potentially identify astrocytic TRPv1 as a novel therapeutic target for treating HIE in the subacute stages.
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USP14 inhibitor attenuates cerebral ischemia/reperfusion-induced neuronal injury in mice.
TL;DR: It is found that IU1 treatment attenuated ischemic stroke‐caused neuronal injury, which was reflected by increased survival rate, reduced infarct volume, as well as decreased neuronal loss in the IU1‐treated mice compared to the control‐ treated mice.
Journal ArticleDOI
TRPV1 promotes repetitive febrile seizures by pro-inflammatory cytokines in immature brain.
Wen-Xian Huang,Fang Yu,Russell M. Sanchez,Yu-Qiang Liu,Jia-Wei Min,Jiang-Jian Hu,Najeeb Bassam Bsoul,Song Han,Jun Yin,Wanhong Liu,Xiaohua He,Biwen Peng +11 more
TL;DR: A critical role is reported for TRPV1 in a febrile seizure mouse model and increased levels of pro-inflammatory factors in the immature brain are revealed, indicating that preventative strategies might be developed for use during febRIle seizures.