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Joel C. Bornstein

Researcher at University of Melbourne

Publications -  246
Citations -  11808

Joel C. Bornstein is an academic researcher from University of Melbourne. The author has contributed to research in topics: Enteric nervous system & Myenteric plexus. The author has an hindex of 57, co-authored 233 publications receiving 10655 citations. Previous affiliations of Joel C. Bornstein include Monash University, Clayton campus & Massachusetts Institute of Technology.

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Alzheimer's disease and Abeta toxicity: from top to bottom.

TL;DR: It is argued that a more integrated, top–down approach to brain function is needed to assess the role of A β in Alzheimer's disease, and that more attention should be paid to the effects of Aβ on synaptic function rather than on cell death.
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Intrinsic primary afferent neurons of the intestine

TL;DR: The intrinsic primaryAfferent neurons of the intestine are the only vertebrate primary afferent neurons so far identified with cell bodies in a peripheral organ and communicate with each other via slow excitatory synaptic potentials in self reinforcing networks and with interneurons and motor neurons via both fast and slow EPSPs.
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Projections and chemical coding of neurons with immunoreactivity for nitric oxide synthase in the guinea-pig small intestine

TL;DR: It is concluded that nitric oxide synthase is located in a sub-population of enteric neurons, amongst which are inhibitory motor neurons that supply the circular muscle layer.
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Enteric motor and interneuronal circuits controlling motility

TL;DR: This review summarizes the knowledge of motor neurones and interneurones in simple motility reflex pathways (ascending and descending excitation, descending inhibition) and it focuses on guinea‐pig ileum.
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Analysis of the responses of myenteric neurons in the small intestine to chemical stimulation of the mucosa

TL;DR: It is concluded that myenteric AH neurons of the guinea pig distal ileum are primary afferent neurons that respond to a variety of mucosally applied chemical stimuli with burst of AP, and the physiologically evoked transmission of slow EPSP to AH neurons suggests that primaryAfferent neurons interconnect in a self-reinforcing network.