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Joel M. Gelfand

Researcher at University of Pennsylvania

Publications -  353
Citations -  26733

Joel M. Gelfand is an academic researcher from University of Pennsylvania. The author has contributed to research in topics: Psoriasis & Population. The author has an hindex of 73, co-authored 325 publications receiving 22107 citations. Previous affiliations of Joel M. Gelfand include Tufts University & Icahn School of Medicine at Mount Sinai.

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Journal ArticleDOI

Risk of myocardial infarction in patients with psoriasis.

Joel M. Gelfand, +5 more
- 11 Oct 2006 - 
TL;DR: Psoriasis may confer an independent risk of MI when controlling for major cardiovascular risk factors, and was greatest in young patients with severe psoriasis, which had an increased adjusted relative risk (RR) for MI that varied by age.
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Prevalence of cardiovascular risk factors in patients with psoriasis

Andrea L. Neimann, +5 more
- 01 Nov 2006 - 
TL;DR: Cardiovascular risk factors that are key components of the metabolic syndrome are more strongly associated with severe psoriasis than with mild Psoriasis.
Journal ArticleDOI

The Risk of Depression, Anxiety, and Suicidality in Patients With Psoriasis: A Population-Based Cohort Study

Shanu Kohli Kurd, +3 more
- 16 Aug 2010 - 
TL;DR: Patients with psoriasis have an increased risk of depression, anxiety, and suicidality according to a population-based cohort study using data collected as part of patient's electronic medical record from 1987 to 2002.
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Psoriasis and comorbid diseases: Epidemiology.

Junko Takeshita, +6 more
- 01 Mar 2017 - 
TL;DR: A rapidly expanding body of literature in various populations and settings supports additional associations between psoriasis and cardiometabolic diseases, gastrointestinal diseases, kidney disease, malignancy, infection, and mood disorders.
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Patients with severe psoriasis are at increased risk of cardiovascular mortality: cohort study using the General Practice Research Database

Nehal N. Mehta, +5 more
- 01 Apr 2010 - 
TL;DR: Patients with severe psoriasis have an increased risk of CV mortality that is independent of traditional CV risk factors, and the findings were robust to multiple sensitivity analyses.