Julian I. Schroeder

TL;DR: PYR/PYLs are ABA receptors functioning at the apex of a negative regulatory pathway that controls ABA signaling by inhibiting PP2Cs, illustrating the power of the chemical genetic approach for sidestepping genetic redundancy.

TL;DR: Activation of Ca2+-permeable channels in the plasma membrane of Arabidopsis guard cells by hydrogen peroxide indicates that ABA-induced H2O2 production and the H 2O 2-activated Ca2-activated channels are important mechanisms for A BA-induced stomatal closing.

TL;DR: Identification of two partially redundant Arabidopsis guard cell‐expressed NADPH oxidase catalytic subunit genes, AtrbohD and atrbohF, are reported, providing direct molecular genetic and cell biological evidence that ROS are rate‐limiting second messengers in ABA signaling, and that the AtrbOHD and AtrabohF NADPH oxidationases function in guard cell ABA signal transduction.

TL;DR: The understanding of the core salt-tolerance mechanisms in plants is reviewed and key Na+ transport and detoxification pathways and the impact of epigenetic chromatin modifications on salinity tolerance are reviewed.

TL;DR: Progress in identification of early stomatal signaling components are reviewed, including ABA receptors and CO(2)-binding response proteins, as well as systems approaches that advance the understanding of guard cell-signaling mechanisms.