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Julián Pérez-Pérez

Researcher at Spanish National Research Council

Publications -  30
Citations -  2707

Julián Pérez-Pérez is an academic researcher from Spanish National Research Council. The author has contributed to research in topics: Gene & Dementia. The author has an hindex of 16, co-authored 27 publications receiving 2337 citations. Previous affiliations of Julián Pérez-Pérez include Autonomous University of Madrid.

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ABA is an essential signal for plant resistance to pathogens affecting JA biosynthesis and the activation of defenses in Arabidopsis

TL;DR: The results support a model for ABA affecting JA biosynthesis in the activation of defenses against this oomycete, and reveal an unexpected overrepresentation of ABA response elements in promoters of P. irregulare–responsive genes.
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A Central Regulatory System Largely Controls Transcriptional Activation and Repression Responses to Phosphate Starvation in Arabidopsis

TL;DR: This paper showed that PHR1 and PHL1 are partially redundant transcription factors acting as central integrators of starvation responses, both specific and generic, and they indicate that transcriptional repression responses are an integral part of adaptive responses to stress.
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Sequencing of diverse mandarin, pummelo and orange genomes reveals complex history of admixture during citrus domestication

TL;DR: This work sequence and compare citrus genomes—a high-quality reference haploid clementine genome and mandarin, pummelo, sweet-orange and sour-orange genomes—and shows that cultivated types derive from two progenitor species.
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Improved protein‐binding microarrays for the identification of DNA‐binding specificities of transcription factors

TL;DR: The development of a protein-binding microarray (PBM11) containing all possible double-stranded 11-mers provides a straightforward method for the prediction of biologically active cis-elements, and thus for identification of in vivo DNA targets of TFs.
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C9ORF72 hexanucleotide expansions of 20–22 repeats are associated with frontotemporal deterioration

TL;DR: Describing these families suggests that short C9ORF72 hexanucleotide expansions are also related to frontotemporal cognitive deterioration, and clear segregation of the 20–22 repeats allele with the disease.