Juliana Madureira de Souza

Bio: Juliana Madureira de Souza is an academic researcher. The author has contributed to research in topics: Periodontitis & Liquid diet. The author has co-authored 2 publications.

NORMATIZAÇÃO DO SUS E PASTORAL DA CRIANÇA QUANTO À DOENÇA PERIODONTAL E CONDIÇÃO PERIODONTAL DAS GESTANTES ACOMPANHADAS Brasilian Plubic Health Care Sistem (SUS) and Children Pastoral Care normatizations for periodontal disease and evaluation of the attended pregnant periodontal conditions

Abstract: The health professionals and Assistant Institutions usually give priority to attend pregnant during the period previous to birth, securing to pregnant woman necessarily care to her baby be born healthful. However it is noticed that actions or activities are limited and don’t inform the impact of periodontal disease has on the prematurity and the low birth weight. This study evaluated the normative judgment of “Pastoral da Crianca” (religion organization that gives children pastoral care) and “Sistema Unico de Saude” (public health care, Brazil) and registered by means of questionnaire and evaluation of periodontal condition in pregnant in “Pastoral da Crianca” of Pindamonhangaba-SP, Brazil, how much the presence of the periodontal disease and orientation received about the risk of periodontitis in pregnancy. The unfamiliarity was observed, on the part of the pregnant with periodontitis, on the risk of the periodontal disease in the gestation. The pregnant deserves special attention; a time that moderate to severe periodontitis disease is a factor of risk for the birth of premature babies or with low weight. UNITERMS: pregnancy; mortality; prematurity; periodontitis; risk factor; periodontitis; prenatal care. R. Periodontia 18(3):57-62 62 REFERENCIAS BIBLIOGRAFICAS 1Paquette DW. The concept of “risk” and emerging discipline of periodontal medicine. J Comtemporary D Practice 1999;1(1):1-18. 2Offenbacher S, Boggess KA, Murtha AP, Jared HL, Lieff S, Mckaig RG et al. Progressive Periodontal disease and risk of very preterm delivery. Obstetrics & Gynecology 2006;107:29-36. 3Boggess KA. Pathophysiology of preterm birth: emerging concepts of maternal infection. Clin Perinatol 2005;32:561-9. 4Zina LG, Moimaz SAS, Saliba NA, Garbin CAS. Periodontite materna e parto prematuro: aspectos biologicos, epidemiologicos e preventivos. Rev Periodontia 2005;15(03):10-5. 5Cruz SS, Costa MCN, Gomes Filho IS, Vianna MIP, Santos CT. Doenca periodontal materna como fator associado ao baixo peso ao nascer. Rev Saude Publica 2005;39(5):782-7. 6Yeo BK, Lim LP, Paquette DW, Williams RC. Periodontal disease the emergence of a risk for systemic conditions: pre-term low birth weight. Ann Acad Med Singapore 2005;34(1):111-6. 7Molitermo LF, Monteiro B, Figueredo CM, Fischer RG. Association between periodontitis and low birth weight: a case-control study. J Clin Periodontol 2005;32(8):886-90. 8Pizzo G, LA Cara M, Conti Nibaldi M, Guiglia R. Periodontitis and preterm delivery. A review of the literature. Minerva Stomatol 2005;54(1-2):1-

Periodontal inflammation induced by chronic ethanol consumption in ovariectomized rats Inflamação periodontal induzida pelo consumo crônico do etanol em ratas ovariectomizadas

Abstract: The immune system plays an important role in the pathogenesis of periodontal diseases.The host may modulate periodontal inflammatory reactions and it determines variances in the individual susceptibility and in the periodontal disease progression speed. Osteoporosis and alcoholism are described as risk indicators of periodontal disease among the systemic acquired factors. Objective: The current study aims to analyze chronic alcohol consumption influence on induced periodontitis in rats presenting estrogen deficiency. Material and Methods: Sixty rats approximately 90 days old were used in the experiment; they were divided into two groups: correlated surgery (OVZ) or surgical ovariectomy simulation (SHAM). Each group was divided into three subgroups: (C) control diet, (A) ethanol containing 20% liquid diet and (I) par-fed control diet. Thirty days after castration the diet and the experimental periodontitis induction were kept for 56 days. Interproximal regions between the first and the second lower left molar and the respective contralateral site without periodontal disease induction were assessed for inflammatory features. Results: Hormone deficiency resulted in important inflammatory changes concerning the meaning of SHAM-C and OVZ-C. The ethanol diet has resulted in inflammatory changes to both groupsSHAM-A and OVZ-A in the absence of periodontitis, with also greater severity when combined with ovariectomy. Conclusion: It was concluded that the association between estrogen deficiency and 20% ethanol was just relevant for sites without periodontitis disease induction, since it induces stronger severity in the inflammatory process in the presence of the