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Julio Acosta-Cabronero

Researcher at UCL Institute of Neurology

Publications -  74
Citations -  4897

Julio Acosta-Cabronero is an academic researcher from UCL Institute of Neurology. The author has contributed to research in topics: Quantitative susceptibility mapping & Diffusion MRI. The author has an hindex of 32, co-authored 72 publications receiving 3919 citations. Previous affiliations of Julio Acosta-Cabronero include University of Cambridge & University College London.

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What the left and right anterior fusiform gyri tell us about semantic memory

TL;DR: The best, indeed the only, strong predictor of semantic scores across a set of 21 patients with frontotemporal lobar degeneration with semantic impairment was degree of hypometabolism in the anterior fusiform region subjacent to the head and body of the hippocampus.
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Absolute diffusivities define the landscape of white matter degeneration in Alzheimer's disease.

TL;DR: It is found that increased absolute diffusivities in Alzheimer's disease were concordant in a distribution consistent with the network hypothesis, highly statistically significant and far more sensitive than fractional anisotropy reductions.
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In Vivo Quantitative Susceptibility Mapping (QSM) in Alzheimer's Disease

TL;DR: The regional and whole-brain cross-sectional comparisons between Alzheimer's disease subjects and matched controls indicate that there may be significant magnetic susceptibility differences for deep brain nuclei – particularly the putamen – as well as for posterior grey and white matter regions.
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Understanding social dysfunction in the behavioural variant of frontotemporal dementia : the role of emotion and sarcasm processing

TL;DR: It was shown that the sarcasm (and emotion recognition) deficit was dependent on a circuit involving the lateral orbitofrontal cortex, insula, amygdala and temporal pole, particularly on the right, and this appeared to be a major factor underlying the deficit in sarcasm recognition.
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Semantic dementia and fluent primary progressive aphasia: two sides of the same coin?

TL;DR: These findings support a claim that the patients' deficits on both verbal and non-verbal tasks reflect progressive deterioration of an amodal integrative semantic memory system critically involving the rostral temporal lobes, rather than a combination of atrophy in the left language network and a separate bilateral ventrotemporal-fusiform network.