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Kai Kaarniranta

Researcher at University of Eastern Finland

Publications -  380
Citations -  30758

Kai Kaarniranta is an academic researcher from University of Eastern Finland. The author has contributed to research in topics: Retinal pigment epithelium & Autophagy. The author has an hindex of 67, co-authored 329 publications receiving 24913 citations. Previous affiliations of Kai Kaarniranta include Åbo Akademi University & Oulu University Hospital.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Antagonistic crosstalk between NF-κB and SIRT1 in the regulation of inflammation and metabolic disorders.

Anu Kauppinen, +4 more
- 01 Oct 2013 - 
TL;DR: The molecular mechanisms of the antagonistic signaling between NF-κB and SIRT1 are examined and how this crosstalk controls inflammatory process and energy metabolism is described to induce the appearance of chronic inflammation in metabolic diseases.
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AMP-activated protein kinase inhibits NF-κB signaling and inflammation: impact on healthspan and lifespan.

Antero Salminen, +2 more
- 23 Mar 2011 - 
TL;DR: The AMPK pathways involved in the inhibition of NF-κB signaling and suppression of inflammation are reviewed and it is emphasized that the capacity of AMPK to repress inflammatory responses can have a significant impact on both healthspan and lifespan.