K
Kavita Babu
Researcher at Indian Institute of Science
Publications - 31
Citations - 406
Kavita Babu is an academic researcher from Indian Institute of Science. The author has contributed to research in topics: Caenorhabditis elegans & Synapse. The author has an hindex of 9, co-authored 28 publications receiving 304 citations. Previous affiliations of Kavita Babu include National University of Singapore & Institute of Molecular and Cell Biology.
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Journal ArticleDOI
A neuropeptide-mediated stretch response links muscle contraction to changes in neurotransmitter release.
Zhitao Hu,Edward C.G. Pym,Kavita Babu,Amy B. Vashlishan Murray,Amy B. Vashlishan Murray,Joshua M. Kaplan +5 more
TL;DR: It is suggested that NLP-12 mediates a mechanosensory feedback loop that couples muscle contraction to changes in presynaptic release, thereby providing a mechanism for proprioceptive control of locomotion.
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Roles of Bifocal, Homer, and F-actin in anchoring Oskar to the posterior cortex of Drosophila oocytes
TL;DR: It is shown that Homer and Bifocal act redundantly to promote posterior anchoring of the osk gene products, and the data suggest that two processes, one requiring B ifocal and an intact F-actin cytoskeleton and a second requiring Homer but independent of intact F -actin, may act redundant to mediate posterior anchored of the Osk gene Products.
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The Immunoglobulin Super Family Protein RIG-3 Prevents Synaptic Potentiation and Regulates Wnt Signaling
TL;DR: The results identify RIG-3 as a regulator of Wnt signaling, and suggest that Rig-3 has an anti-plasticity function that prevents activity-induced changes in postsynaptic receptor fields.
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ATP-dependent membrane remodeling links EHD1 functions to endocytic recycling.
Raunaq Deo,Manish S. Kushwah,Sukrut C. Kamerkar,Nagesh Y. Kadam,Srishti Dar,Kavita Babu,Anand Srivastava,Thomas J. Pucadyil +7 more
TL;DR: It is reported that EHD1 hydrolyzes ATP to remodel, causing fission of membrane tubes and that this is necessary for endocytic recycling, and in vitro reconstitution assays and molecular dynamics simulations corroborate this scission pathway.
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FLP-18 Functions through the G-Protein-Coupled Receptors NPR-1 and NPR-4 to Modulate Reversal Length in Caenorhabditis elegans
TL;DR: The circuit and molecular machinery required for normal reversal behavior in hermaphrodite Caenorhabditis elegans is elucidated, and it is shown that FLP-18 functions to maintain reversal length through the neuropeptide receptors NPR-4 and NPR-1.