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Kodiveri Muthukaliannan Gothandam

Researcher at VIT University

Publications -  88
Citations -  1564

Kodiveri Muthukaliannan Gothandam is an academic researcher from VIT University. The author has contributed to research in topics: Gene & Publication bias. The author has an hindex of 19, co-authored 78 publications receiving 1108 citations. Previous affiliations of Kodiveri Muthukaliannan Gothandam include Chungbuk National University & C. Abdul Hakeem College.

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OsPPR1, a pentatricopeptide repeat protein of rice is essential for the chloroplast biogenesis.

TL;DR: The results suggest that the OsPPR1 is a nuclear gene of rice, encoding the PPR protein that might play a role in the chloroplast biogenesis.
Journal Article

The effect of salinity on growth, hormones and mineral elements in leaf and fruit of tomato cultivar PKM1.

TL;DR: Results showed that leaf area and dry matter content of tomato fruits decreased with application of elevated salt stress, however endogenous content of IAA, ABA and proline was found to be increasing with increase in salt treatment, suggesting that leaves are more sensitive than fruits.
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Prognostic significance of blood inflammatory biomarkers NLR, PLR, and LMR in cancer-A protocol for systematic review and meta-analysis.

TL;DR: This meta-analysis protocol examines the comparative utility of neutrophil-lymphocyte-ratio, platelet-ly mphocyte- ratio, and monocyte-lyymphocyte-Ratio as a simple, inexpensive, and effective method for cancer prognosis, in multiple cancers.
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Screening and optimization of protease production from a halotolerant Bacillus licheniformis isolated from saltern sediments

TL;DR: Studies on the effect of different carbon and nitrogen sources revealed that xylose and urea enhances the enzyme production, and with selected C–N sources along with 1 M NaCl the maximum protease production was obtained.
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Phloretin loaded chitosan nanoparticles augments the pH-dependent mitochondrial-mediated intrinsic apoptosis in human oral cancer cells.

TL;DR: The findings suggested that PhCsNPs augmented the mitochondrial-mediated apoptotic mechanism through the stimulation of oxidative stress, depletion of cellular antioxidants and cell cycle arrest, and could be used as an efficient therapeutic agent for the treatment of oral cancer.