K
Konrad Messmer
Researcher at Ludwig Maximilian University of Munich
Publications - 145
Citations - 8214
Konrad Messmer is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topics: Microcirculation & Reperfusion injury. The author has an hindex of 44, co-authored 145 publications receiving 8000 citations. Previous affiliations of Konrad Messmer include University of Münster & University of Mainz.
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Journal ArticleDOI
Incidence and severity of anaphylactoid reactions to colloid volume substitutes
Johannes Ring,Konrad Messmer +1 more
TL;DR: All available colloid volume substitutes carry the risk of anaphylactoid reactions, but the frequency of severe reactions was low for plasma-protein solutions, hydroxyethyl starch, 0-008% for dextran, and 0-038% for gelatin solutions.
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Orthogonal polarization spectral imaging: a new method for study of the microcirculation.
Warren Groner,James W. Winkelman,A. G. Harris,Can Ince,Gerrit J. Bouma,Konrad Messmer,Richard G. Nadeau +6 more
TL;DR: Orthogonal polarization spectral (OPS) imaging is reported here on, a new method for imaging the microcirculation using reflected light that allows imaging of the micro Circulation noninvasively through mucus membranes and on the surface of solid organs.
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Platelet-endothelial cell interactions during ischemia/reperfusion : The role of P-selectin
Steffen Massberg,Georg Enders,Georg Enders,Rosmarie Leiderer,Rosmarie Leiderer,Simone Eisenmenger,Simone Eisenmenger,Dietmar Vestweber,Dietmar Vestweber,Fritz Krombach,Fritz Krombach,Konrad Messmer,Konrad Messmer +12 more
TL;DR: First in vivo evidence is provided that platelets accumulate in the postischemic microvasculature early after reperfusion via P-selectin-ligand interactions, indicating that platelet recruitment and subsequent activation might play an important role in the pathogenesis of I/R injury.
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The beneficial effect of human recombinant superoxide dismutase on acute and chronic rejection events in recipients of cadaveric renal transplants.
Walter Land,Helmut Schneeberger,Stefan Schleibner,Wolf-Dieter Illner,D. Abendroth,Gianfranco Rutili,Karl E. Arfors,Konrad Messmer +7 more
TL;DR: Rh-SOD has the potential to mitigate free radical-mediated reperfusion injury-induced acute endothelial cell damage that potentially may contribute to the process of chronic obliterative rejection arteriosclerosis and in accordance with the "response-to-injury hypothesis" in the pathogenesis of general atherosclerosis.
Journal ArticleDOI
Microvascular ischemia-reperfusion injury in striated muscle: significance of "reflow paradox"
TL;DR: The concept that reactive oxygen metabolites contribute to I/R-induced reflow paradox, resulting in leukocyte accumulation, adherence, and increase in microvascular permeability is supported.