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Krisztina Takács-Vellai

Researcher at Eötvös Loránd University

Publications -  41
Citations -  12193

Krisztina Takács-Vellai is an academic researcher from Eötvös Loránd University. The author has contributed to research in topics: Caenorhabditis elegans & Autophagy. The author has an hindex of 23, co-authored 40 publications receiving 10613 citations. Previous affiliations of Krisztina Takács-Vellai include University of Fribourg.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Genetics: Influence of TOR kinase on lifespan in C. elegans

Tibor Vellai, +5 more
- 11 Dec 2003 - 
TL;DR: It is shown that TOR deficiency in the nematode Caenorhabditis elegans more than doubles its natural lifespan, suggesting a new function for TOR signalling in ageing control may represent a link between nutrition, metabolism and longevity.
Journal ArticleDOI

Longevity pathways converge on autophagy genes to regulate life span in Caenorhabditis elegans.

Marton L. Toth, +10 more
- 30 Jan 2008 - 
TL;DR: It is demonstrated that mutational inactivation of autophagy genes, which are involved in the degradation of aberrant, damaged cytoplasmic constituents accumulating in all aging cells, accelerates the rate at which the tissues age in the nematode Caenorhabditis elegans and may act as a central regulatory mechanism of animal aging.
Journal ArticleDOI

Inactivation of the autophagy gene bec-1 triggers apoptotic cell death in C. elegans.

Krisztina Takács-Vellai, +7 more
- 23 Aug 2005 - 
TL;DR: It is proposed that bec-1 represents a link between autophagy and apoptosis, thus supporting the view that the two processes act in concerted manner in the cell death machinery.