L
Leona D. Samson
Researcher at Massachusetts Institute of Technology
Publications - 214
Citations - 20417
Leona D. Samson is an academic researcher from Massachusetts Institute of Technology. The author has contributed to research in topics: DNA repair & DNA damage. The author has an hindex of 70, co-authored 212 publications receiving 18227 citations. Previous affiliations of Leona D. Samson include University of California, Berkeley & Harvard University.
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The Lancet Commission on pollution and health
Philip J. Landrigan,Richard Fuller,Nereus J R Acosta,Olusoji Adeyi,Robert G. Arnold,Niladri Basu,Abdoulaye Bibi Baldé,Roberto Bertollini,Stephan Bose-O'Reilly,Stephan Bose-O'Reilly,Jo Ivey Boufford,Patrick N. Breysse,Thomas C. Chiles,Chulabhorn Mahidol,Awa M Coll-Seck,Maureen L. Cropper,Julius N. Fobil,Valentin Fuster,Valentin Fuster,Michael Greenstone,Andy Haines,David Hanrahan,David J. Hunter,Mukesh Khare,Alan Krupnick,Bruce P. Lanphear,Bindu Lohani,Keith Martin,Karen V Mathiasen,Maureen A. McTeer,Christopher J L Murray,Johanita D Ndahimananjara,Frederica P. Perera,Janez Potočnik,Alexander S Preker,Alexander S Preker,Jairam Ramesh,Johan Rockström,Carlos Salinas,Leona D. Samson,Karti Sandilya,Peter D. Sly,Kirk R. Smith,Achim Steiner,Richard B. Stewart,William A. Suk,Onno C. P. van Schayck,Gautam N. Yadama,Kandeh K. Yumkella,Ma Zhong +49 more
TL;DR: This book is dedicated to the memory of those who have served in the armed forces and their families during the conflicts of the twentieth century.
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Systematic Discovery of In Vivo Phosphorylation Networks
Rune Linding,Rune Linding,Lars Juhl Jensen,Gerard J. Ostheimer,Marcel A. T. M. van Vugt,Marcel A. T. M. van Vugt,Claus Jørgensen,Ioana M. Miron,Francesca Diella,Karen Colwill,Lorne Taylor,Kelly Elder,Pavel Metalnikov,Vivian Nguyen,Adrian Pasculescu,Jing Jin,Jin Park,Leona D. Samson,James R. Woodgett,Robert B. Russell,Peer Bork,Michael B. Yaffe,Tony Pawson +22 more
TL;DR: An approach is developed that augments motif-based predictions with the network context of kinases and phosphoproteins, which suggests that BCLAF1 is a GSK-3 substrate and yields a 2.5-fold improvement in the accuracy with which phosphorylation networks can be constructed.
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Balancing repair and tolerance of DNA damage caused by alkylating agents
TL;DR: In this article, the response of an individual to alkylating agents can vary considerably from tissue to tissue and from person to person, pointing to genetic and epigenetic mechanisms that modulate alkylation agent toxicity.
Journal ArticleDOI
A new pathway for DNA repair in Escherichia coli.
Leona D. Samson,John Cairns +1 more
TL;DR: The relationship between ambient concentration of various mutagens and resulting mutation rate was determined and the first mutagen studied was MNNG and the results were totally unexpected.
Balancing repair and tolerance of DNA damage caused by alkylating agents
TL;DR: Alkylating agents constitute a major class of frontline chemotherapeutic drugs that inflict cytotoxic DNA damage as their main mode of action, in addition to collateral mutagenic damage, and the response of an individual to alkylates agents can vary considerably from tissue to tissue and from person to person.