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Lijun Jia

Researcher at Shanghai University

Publications -  81
Citations -  8885

Lijun Jia is an academic researcher from Shanghai University. The author has contributed to research in topics: Neddylation & Ubiquitin ligase. The author has an hindex of 26, co-authored 69 publications receiving 7059 citations. Previous affiliations of Lijun Jia include Fudan University & University of Michigan.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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The Nedd8-activating enzyme inhibitor MLN4924 induces autophagy and apoptosis to suppress liver cancer cell growth.

Zhongguang Luo, +16 more
- 01 Jul 2012 - 
TL;DR: It is reported that MLN4924 also triggers autophagy in response to CRL inactivation and that this effect is important for the ability of MLN 4924 to suppress the outgrowth of liver cancer cells in vitro and in vivo.
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Overactivated Neddylation Pathway as a Therapeutic Target in Lung Cancer

Lihui Li, +28 more
- 01 Jun 2014 - 
TL;DR: The overactivated neddylation pathway in lung cancer development and as a promising therapeutic target is highlighted in both lung adenocarcinoma and squamous-cell carcinoma.
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Protein neddylation and its alterations in human cancers for targeted therapy.

Lisha Zhou, +4 more
- 01 Apr 2018 - 
TL;DR: Recently, increasing pieces of experimental evidence strongly indicate that the process of protein neddylation modification is elevated in multiple human cancers, providing sound rationale for its targeting as an attractive anticancer therapeutic strategy.