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Lloyd A. Greene

Researcher at Columbia University

Publications -  259
Citations -  40969

Lloyd A. Greene is an academic researcher from Columbia University. The author has contributed to research in topics: Nerve growth factor & Neurite. The author has an hindex of 102, co-authored 257 publications receiving 37663 citations. Previous affiliations of Lloyd A. Greene include New York University & Boston Children's Hospital.

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Establishment of a noradrenergic clonal line of rat adrenal pheochromocytoma cells which respond to nerve growth factor.

TL;DR: A single cell clonal line which responds reversibly to nerve growth factor (NGF) has been established from a transplantable rat adrenal pheochromocytoma and should be a useful model system for neurobiological and neurochemical studies.
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Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.

Lorenzo Galluzzi, +186 more
TL;DR: The Nomenclature Committee on Cell Death (NCCD) has formulated guidelines for the definition and interpretation of cell death from morphological, biochemical, and functional perspectives.
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Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018

Lorenzo Galluzzi, +168 more
- 01 Jan 2018 - 
TL;DR: An updated classification of cell death subroutines focusing on mechanistic and essential aspects of the process is proposed, and the utility of neologisms that refer to highly specialized instances of these processes are discussed.
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Nerve growth factor and epidermal growth factor induce rapid transient changes in proto-oncogene transcription in PC12 cells.

TL;DR: The results suggest that c-fos, c-myc, and actin induction may be general nuclear responses to growth or differentiation factors in a variety of different cell types.
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Stimulation of neuronal acetylcholine receptors induces rapid gene transcription

TL;DR: It is suggested that neurotransmitters may rapidly activate specific gene transcription in nondividing neuronally differentiated cells and a functional role for neurotransmitter induction of c-fos and actin expression in the nervous system is suggested.