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Luis M. Ruilope

Researcher at European University of Madrid

Publications -  891
Citations -  109166

Luis M. Ruilope is an academic researcher from European University of Madrid. The author has contributed to research in topics: Blood pressure & Renal function. The author has an hindex of 94, co-authored 841 publications receiving 97778 citations. Previous affiliations of Luis M. Ruilope include Lund University & Mayo Clinic.

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Effect of finerenone on ambulatory blood pressure in chronic kidney disease in type 2 diabetes

TL;DR: In this article , the effects of finerenone on 24-hour ambulatory BP in patients with chronic kidney disease and type 2 diabetes were evaluated using ARTS-DN (NCT01874431) clinical trial.
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Therapeutic implications and new perspectives for essential hypertension and renal damage

TL;DR: The need to explore the renoprotective and cardiovascular protective capacity of the different classes of antihypertensive drugs, in patients with essential hypertension and some degree of renal involvement, characterized by the presence of microalbuminuria, proteinuria and/or an elevated serum creatinine is explored.
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Renal effects of amino acid infusions in patients with panhypopituitarism.

TL;DR: The results do not support involvement of the pituitary gland in the acute renal response to amino acids in patients with panhypopituitarism.
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Efficacy and tolerability of a single-pill combination of telmisartan 80 mg and hydrochlorothiazide 25 mg according to age, gender, race, hypertension severity, and previous antihypertensive use: planned analyses of a randomized trial.

TL;DR: The T80/H25 single-pill combination provides consistent BP reductions and higher goal attainment rates versus T80 across a range of hypertensive patient subgroups, which is likely to have a positive impact on patients’ cardiovascular risk.
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Evaluation of the renal effects of calcium antagonists.

TL;DR: Calcium antagonists can improve the short‐ and long‐term prognosis for renal function in human transplantation through their effects in avoiding cyclosporine‐induced renal vasoconstriction and in facilitating renal sodium output.