L
Luis M. Ruilope
Researcher at European University of Madrid
Publications - 891
Citations - 109166
Luis M. Ruilope is an academic researcher from European University of Madrid. The author has contributed to research in topics: Blood pressure & Renal function. The author has an hindex of 94, co-authored 841 publications receiving 97778 citations. Previous affiliations of Luis M. Ruilope include Lund University & Mayo Clinic.
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Evidence of chronic kidney injury in patients not meeting KDIGO criteria for chronic kidney disease
TL;DR: Tools to predict albuminuria development and to identify those subjects who will benefit most from intervention are limited, but the incidence of subclinical atherosclerosis, cardiovascular events and CKD progression is already increased in the high-normal UACR range.
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Response to Out-of-Office Blood Pressure Control Among Treated Subjects
TL;DR: In this study on 12 897 treated patients with hypertension in a primary care setting in Spain, office systolic blood pressure (BP)/diastolic …
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Influence of chronic kidney disease development and renin-angiotensin system inhibition on cardiovascular prognosis.
TL;DR: The high prevalence of chronic kidney disease in the general and in the hypertensive populations forces the recognition of its relevance and the need for an integrative therapeutic approach to protect simultaneously renal and cardiovascular systems.
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TCT-84 Renal Denervation With Symplicity Spyral Catheter in the Global Symplicity Registry: Safety and Efficacy at 3 Years
Michael Böhm,Felix Mahfoud,Bryan Williams,Luis M. Ruilope,Krzysztof Narkiewicz,Markus P. Schlaich,Martin Fahy,Giuseppe Mancia +7 more
TL;DR: The current analysis presents data on safety and efficacy outcomes up to 3 years from the GSR, a prospective, international registry of patients treated with the Symplicity Spyral catheter.
Journal Article
Aldosterone regulation in anephric patients.
TL;DR: ACTH and potassium originate a significant increase in plasma aldosterone, and seem to be the main ald testosterone secretion regulators in the absence of renin production.