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Maria Tiziana Corasaniti

Researcher at Magna Græcia University

Publications -  156
Citations -  12024

Maria Tiziana Corasaniti is an academic researcher from Magna Græcia University. The author has contributed to research in topics: Neuroprotection & Medicine. The author has an hindex of 43, co-authored 138 publications receiving 9824 citations. Previous affiliations of Maria Tiziana Corasaniti include University of Pavia & Sapienza University of Rome.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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S -nitrosylation regulates apoptosis

TL;DR: It is shown that NO-mediated S-nitrosylation of the cysteine-containing enzymes that mediate apoptosis (caspases and tissue-transglutaminase, tTG) regulates the balance between apoptosis and necrosis.
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Post‐ischemic brain damage: pathophysiology and role of inflammatory mediators

TL;DR: The present work reviewed the role of neuroinflammatory mediators in the pathophysiology of ischemic brain damage and their potential exploitation as drug targets for the treatment of cerebral ischemia.
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From clinical evidence to molecular mechanisms underlying neuroprotection afforded by estrogens.

TL;DR: Recent findings concerning the neuronal effects of estrogens that may contribute to their neuroprotective actions are discussed, and both estrogen receptor-dependent and -independent mechanisms will be described.