M
Mark E. Cooper
Researcher at University of Queensland
Publications - 1514
Citations - 141899
Mark E. Cooper is an academic researcher from University of Queensland. The author has contributed to research in topics: Diabetes mellitus & Diabetic nephropathy. The author has an hindex of 158, co-authored 1463 publications receiving 124887 citations. Previous affiliations of Mark E. Cooper include University of Cambridge & University of Adelaide.
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Targeting advanced glycation endproducts and mitochondrial dysfunction in cardiovascular disease
TL;DR: The AGE-RAGE axis may contribute to a proinflammatory environment inducing cellular dysfunction which cascades towards pathology as mentioned in this paper, which is a leading cause of mortality in the Western World.
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Laurentian crustal recycling in the Ordovician Grampian Orogeny: Nd isotopic evidence from western Ireland
TL;DR: In this paper, a detailed Nd isotopic stratigraphy for volcanic and volcaniclastic formations from the South Mayo Trough, an accreted oceanic arc exposed in the western Irish Caledonides, is presented.
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Pro-resolving lipid mediators: regulators of inflammation, metabolism and kidney function.
TL;DR: In this article, the role of endogenous lipid mediators (i.e., specialized pro-resolving lipid mediator and branched fatty acid esters of hydroxy fatty acids) in the resolution of chronic kidney disease (CKD) and kidney failure is discussed.
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Diabetic nephropathy in 2010: Alleviating the burden of diabetic nephropathy.
Stephen P. Gray,Mark E. Cooper +1 more
TL;DR: Many patients with diabetic nephropathy progress to end-stage renal disease, and new research in disease detection, diagnosis, and novel treatments will hopefully alleviate the burden of diabetic neophropathy in the future.
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The selective values of alleles in a molecular network model are context dependent
TL;DR: A bridge between genetics and gene network theories is provided by relating key concepts from quantitative genetics to the parameters, variables, and performance functions of genetic networks by simulating the genetic switch controlling galactose metabolism in yeast and its response to selection.