M
Mark E. Cooper
Researcher at University of Queensland
Publications - 1514
Citations - 141899
Mark E. Cooper is an academic researcher from University of Queensland. The author has contributed to research in topics: Diabetes mellitus & Diabetic nephropathy. The author has an hindex of 158, co-authored 1463 publications receiving 124887 citations. Previous affiliations of Mark E. Cooper include University of Cambridge & University of Adelaide.
Papers
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Journal ArticleDOI
Epigenetics: Mechanisms and Implications for Diabetic Complications
Mark E. Cooper,Assam El-Osta +1 more
TL;DR: It is postulated that further understanding enzymes involved in writing and erasing chemical changes could transform the understanding of the pathways implicated in diabetic vascular injury providing new therapeutic strategies.
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Up-regulation of components of the renin-angiotensin system in the bile duct–ligated rat liver
G. Paizis,Mark E. Cooper,Josefa M. Schembri,Christos Tikellis,Louise M Burrell,Peter W Angus +5 more
TL;DR: Key elements of the RAS are present in normal liver tissue, and there is major up-regulation of the system in the bile duct-ligated liver.
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Progressive decline in renal function in diabetic patients with and without albuminuria.
Con Tsalamandris,Terri J Allen,Richard E Gilbert,Ashim Sinha,Sianna Panagiotopoulos,Mark E. Cooper,George Jerums +6 more
TL;DR: The concept that a subgroup of diabetic patients may show a decline in renal function in the absence of significant increases in AER is supported and it is suggested that albuminuria alone may not predict renal functional changes in all diabetic patients.
Journal ArticleDOI
Screening for drought resistance in rainfed lowland rice
TL;DR: Addition of a drought screening program that is conducted in the field in the wet-season to the overall breeding program would enhance the opportunity to select for drought resistance within the breeding materials and increase the chance of developing high yielding cultivars adapted to the drought-prone rainfed lowland environments.
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Neutrophils--a key component of ischemia-reperfusion injury.
TL;DR: Inhibiting neutrophil mobilization, tissue recruitment, and ultimately neutrophils-associated activation of local and systemic inflammatory responses may have therapeutic potential in the amelioration ofLocal and remote tissue damage following IRI.