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Mark Hallett

Researcher at National Institutes of Health

Publications -  1234
Citations -  136876

Mark Hallett is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transcranial magnetic stimulation & Motor cortex. The author has an hindex of 186, co-authored 1170 publications receiving 123741 citations. Previous affiliations of Mark Hallett include Government of the United States of America & Armed Forces Institute of Pathology.

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Motor skill learning in patients with cerebellar degeneration

TL;DR: Patients with cerebellar degeneration can exhibit almost normal performance in skill learning with slow movements, but with fast movements, their performance improves to a lesser extent, suggesting difficulty in the refinement of motor execution, which is more of a requirement for fast movements than for slow ones.
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Small de novo duplication in the repeat region of the TATA-box-binding protein gene manifest with a phenotype similar to variant Creutzfeldt-Jakob disease.

TL;DR: The findings suggest that patients suspected of vCJD should undergo testing for SCA17, Huntington's disease and other neurodegener‐ative disorders having phenotypic similarities with vC JD.
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Brain activity during visuomotor behavior triggered by arbitrary and spatially constrained cues: an fMRI study in humans

TL;DR: Brain activity during visually informed movement involving two different types of cues and two different effectors is explored, suggesting functional demarcation of the PMd between its rostral and dorsal parts during visuomotor mapping.
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Extreme task specificity in writer's cramp.

TL;DR: Focal hand dystonia may be task specific, as the case with writer's cramp, and in early stages, task specificity can be so specific that it may be mistaken for a psychogenic movement disorder.
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Treatment of peripheral neuropathies.

TL;DR: There are three general approaches to treatment of peripheral neuropathy: an attempt should be made to reverse the pathophysiological process if its nature can be elucidated, nerve metabolism can be stimulated and regeneration encouraged, and symptomatic therapy can be employed.