M
Mark Hallett
Researcher at National Institutes of Health
Publications - 1234
Citations - 136876
Mark Hallett is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transcranial magnetic stimulation & Motor cortex. The author has an hindex of 186, co-authored 1170 publications receiving 123741 citations. Previous affiliations of Mark Hallett include Government of the United States of America & Armed Forces Institute of Pathology.
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Where does chorea come from? cortical excitability findings challenge classic pathophysiological concepts.
Mark Hallett,Jose A. Obeso +1 more
TL;DR: Cortical excitability in the chorea-dyskinetic and parkinsonian states are exactly opposite of what had been expected out of the prevailing basal ganglia pathophysiological concepts.
Treatment of Essential Tremor with Long-chain Alcohols: Still Experimental or Ready for Prime Time?
TL;DR: Preclinical efficacy data for OA are positive, and human pilot data demonstrated excellent safety as well as efficacy in secondary outcome measures of tremor amplitudes, suggesting that OA may be worth developing as a pharmaceutical.
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Mirror movements or functional tremor masking organic tremor.
TL;DR: Objective physiology and refinement of the current clinical and physiologic tremor evaluation techniques can help reduce misdiagnosis of functional tremor and help identify an underlying organic tremor etiology.
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Orthostatic myoclonus associated with Caspr2 antibodiesAuthor Response
Jay A. van Gerpen,Felix Gövert,J. Eric Ahlskog,Robert Chen,Victor S.C. Fung,Mark Hallett,Günther Deuschl,Frank Leypoldt +7 more
TL;DR: Govert et al., authors of the case report, defend their diagnosis and explain that OM can be present in the arms and can have subtle action myoclonus, and argues that there is no clear evidence to link macrolides to altered dopamine levels or NMDA receptor activity and that Macrolides have poor CSF.
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Patients with Parkinson disease are prone to functional neurological disorders
TL;DR: Until recently the management of Parkinson disease focused almost entirely on the triad of bradykinesia, rigidity and tremor, but it then became apparent that there are many non-motor features as well, and management of these is often as important as the motor symptoms.