M
Mark Hallett
Researcher at National Institutes of Health
Publications - 1234
Citations - 136876
Mark Hallett is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transcranial magnetic stimulation & Motor cortex. The author has an hindex of 186, co-authored 1170 publications receiving 123741 citations. Previous affiliations of Mark Hallett include Government of the United States of America & Armed Forces Institute of Pathology.
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Postexercise depression of motor evoked potentials: a measure of central nervous system fatigue.
J. P. Brasil-Neto,Alvaro Pascual-Leone,J. Valls-Solé,A. Cammarota,Leonardo G. Cohen,Mark Hallett +5 more
TL;DR: In six normal volunteers, including four of the investigators, the amplitudes of motor evoked potentials elicited by transcranial magnetic stimulation were transiently decreased after exercise, indicating fatigue of motor pathways in the central nervous system.
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Contribution of the ipsilateral motor cortex to recovery after chronic stroke
TL;DR: The effects of transcranial magnetic stimulation on motor performance of the paretic hand of chronic stroke patients and healthy controls were evaluated and it was found that stimulation of the intact hemisphere resulted in delayed simple reaction times (RTs) in the contralateral healthy but not in the ipsilateral pareti hand.
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Task-related coherence and task-related spectral power changes during sequential finger movements
Paolo Manganotti,Christian Gerloff,Camilo Toro,Katsuta H,Norihiro Sadato,P. Zhuang,Letizia Leocani,Mark Hallett +7 more
TL;DR: The present results indicate an active intercommunication between bilateral and mesial central and prefrontal regions which becomes more intense with more complex sequential movements.
Journal ArticleDOI
The non-motor syndrome of primary dystonia: clinical and pathophysiological implications.
TL;DR: Growing evidence indicates an important non-motor component to primary dystonia, including abnormalities in sensory and perceptual functions, as well as neuropsychiatric, cognitive and sleep domains, is indicated.
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Single–joint rapid arm movements in normal subjects and in patients with motor disorders
Alfredo Berardelli,Mark Hallett,John C. Rothwell,Rocco Agostino,M. Manfredi,Philip D. Thompson,C. D. Marsden +6 more
TL;DR: The basal ganglia have a role in scaling the size of AG1, reinforcing the voluntary command and inhibiting inappropriate EMG activity, and the cerebellum has arole in timing the voluntary bursts and probably in implementing muscle force phasically.