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Martin Graef

Researcher at University of Cologne

Publications -  31
Citations -  7979

Martin Graef is an academic researcher from University of Cologne. The author has contributed to research in topics: Autophagy & Autophagosome. The author has an hindex of 19, co-authored 28 publications receiving 6285 citations. Previous affiliations of Martin Graef include Max Planck Society & University of California, Davis.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

ER exit sites are physical and functional core autophagosome biogenesis components

TL;DR: ERES function is required for assembly of the autophagy machinery immediately downstream of the Atg1 kinase complex and is associated with formation of autophagosomes at every stage of the process.
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Mitochondria regulate autophagy by conserved signalling pathways

TL;DR: It is shown that the evolutionarily conserved protein kinases Atg1, target of rapamycin kinase complex I, and protein kinase A (PKA) regulate autophagic flux, whereas autophagy gene induction depends solely on PKA.
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Endoplasmic reticulum-associated mitochondria–cortex tether functions in the distribution and inheritance of mitochondria

TL;DR: These findings identify Num1 as a key component of a mitochondria–ER–cortex anchor, which is termed “MECA,” that functions in parallel with mitochondrial dynamics to distribute and position the essential mitochondrial network.