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Martin Schreiber

Researcher at Medical University of Vienna

Publications -  63
Citations -  6439

Martin Schreiber is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Breast cancer & Genotype. The author has an hindex of 34, co-authored 60 publications receiving 6073 citations. Previous affiliations of Martin Schreiber include Research Institute of Molecular Pathology & University of Vienna.

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DeltaEF1 is a transcriptional repressor of E-cadherin and regulates epithelial plasticity in breast cancer cells.

TL;DR: RNA interference-mediated downregulation of deltaEF1 in cancer cells was sufficient to derepress E-cadherin expression and restore cell to cell adhesion, suggesting that deltaEF 1 is a key player in late stage carcinogenesis.
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The transcription factor ZEB1 (δEF1) promotes tumour cell dedifferentiation by repressing master regulators of epithelial polarity

TL;DR: The data show that ZEB1 represents a key player in pathologic EMTs associated with tumour progression, and downregulation in undifferentiated cancer cells by RNA interference was sufficient to upregulate expression of cell polarity genes on the RNA and protein level, to re-establish epithelial features and to impair cell motility in vitro.
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Control of cell cycle progression by c-Jun is p53 dependent

TL;DR: It is demonstrated that fibroblasts derived from c-jun-/- mouse fetuses exhibit a severe proliferation defect and undergo a prolonged crisis before spontaneous immortalization, and a mechanistic link between c-Jun-dependent mitogenic signaling and cell-cycle regulation is established.
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JNK2 is required for efficient T-cell activation and apoptosis but not for normal lymphocyte development

TL;DR: JNK2 functions in a cell-type-specific and stimulus-dependent manner, being required for apoptosis of immature thymocytes induced by anti-CD3 antibody but not for apoptotic induced byAnti-Fas antibody, UVC or dexamethasone, and is not required for activation-induced cell death of mature T cells.