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Mary J. Morrell

Researcher at Imperial College London

Publications -  44
Citations -  1402

Mary J. Morrell is an academic researcher from Imperial College London. The author has contributed to research in topics: Breathing & Sleep apnea. The author has an hindex of 17, co-authored 42 publications receiving 1280 citations. Previous affiliations of Mary J. Morrell include University of Wisconsin-Madison.

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Evaluation of droplet dispersion during non-invasive ventilation, oxygen therapy, nebuliser treatment and chest physiotherapy in clinical practice: implications for management of pandemic influenza and other airborne infections

TL;DR: The findings suggest that health-care workers providing NIV and chest physiotherapy, working within 1 m of an infected patient should have a higher level of respiratory protection, but that infection control measures designed to limit aerosol spread may have less relevance for these procedures.
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The effect of sleep on reflex genioglossus muscle activation by stimuli of negative airway pressure in humans.

TL;DR: It is shown that non‐REM sleep attenuates reflex genioglossus muscle activation by stimuli of negative airway pressure, which may have implications for the pathogenesis of obstructive sleep apnoea.
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Progressive Retropalatal Narrowing Preceding Obstructive Apnea

TL;DR: Results show that expiratory narrowing produced a significant reduction of CSA at end-expiration prior to obstructive apnea.
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Sleep fragmentation, awake blood pressure, and sleep-disordered breathing in a population-based study.

TL;DR: Using data from the Wisconsin Sleep Cohort Study, the hypothesis that sleep fragmentation is associated with elevated awake blood pressure was tested and the SFI was significantly associated with higher levels of awake systolic blood pressure in people with an AHI < 1.
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Effect of induced hypocapnic hypopnea on upper airway patency in humans during NREM sleep.

TL;DR: The reduction in Vmax during hypopnea in subjects with IFL during eupneic control, suggests that reduced drive is associated with increased upper airway compliance in these subjects, andupper airway susceptibility to narrowing/closure is an important determinant of the response to inducedhypopnea during NREM sleep.