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Mary L. Phillips

Researcher at University of Pittsburgh

Publications -  455
Citations -  45030

Mary L. Phillips is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Bipolar disorder & Prefrontal cortex. The author has an hindex of 105, co-authored 422 publications receiving 39995 citations. Previous affiliations of Mary L. Phillips include King's College & University of Amsterdam.

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Neurobiology of emotion perception I: the neural basis of normal emotion perception

TL;DR: It is suggested that the extent to which a stimulus is identified as emotive and is associated with the production of an affective state may be dependent upon levels of activity within these two neural systems.
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Neurobiology of emotion perception II: Implications for major psychiatric disorders

TL;DR: It is suggested that distinct patterns of structural and functional abnormalities in neural systems important for emotion processing are associated with specific symptoms of schizophrenia and bipolar and major depressive disorder.
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A specific neural substrate for perceiving facial expressions of disgust

TL;DR: Functional magnetic resonance imaging was used to examine the neural substrate for perceiving disgust expressions and found the neural response to facial expressions of disgust in others is thus closely related to appraisal of distasteful stimuli.
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A neural model of voluntary and automatic emotion regulation: implications for understanding the pathophysiology and neurodevelopment of bipolar disorder.

TL;DR: A neural model of emotion regulation that includes neural systems implicated in different voluntary and automatic emotion regulatory subprocesses is developed and used as a theoretical framework to examine functional neural abnormalities in these neural systems that may predispose to the development of a major psychiatric disorder, bipolar disorder.
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Distinct Neural Correlates of Washing, Checking, and Hoarding Symptom Dimensions in Obsessive-compulsive Disorder

TL;DR: The findings suggest that different obsessive-compulsive symptom dimensions are mediated by relatively distinct components of frontostriatothalamic circuits implicated in cognitive and emotion processing.