scispace - formally typeset
M

Massimo Nabissi

Researcher at University of Camerino

Publications -  121
Citations -  10470

Massimo Nabissi is an academic researcher from University of Camerino. The author has contributed to research in topics: Transient receptor potential channel & Receptor. The author has an hindex of 37, co-authored 112 publications receiving 8363 citations. Previous affiliations of Massimo Nabissi include University of Western Australia.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Capsaicin-induced apoptosis of glioma cells is mediated by TRPV1 vanilloid receptor and requires p38 MAPK activation

TL;DR: P38 but not extracellular signal‐regulated protein kinase activation was required for TRPV1‐mediated CPS‐induced apoptosis of glioma cells, which was functionally implicated in these events as they were markedly inhibited by the TRpV1 antagonist, capsazepine.
Journal ArticleDOI

Triggering of the TRPV2 channel by cannabidiol sensitizes glioblastoma cells to cytotoxic chemotherapeutic agents

TL;DR: It is demonstrated that co-administration of cytotoxic agents together with the TRPV2 agonist CBD increases drug uptake and parallelly potentiates cytot toxic activity in human glioma cells.