M
Maurizio Cammalleri
Researcher at University of Pisa
Publications - 71
Citations - 1817
Maurizio Cammalleri is an academic researcher from University of Pisa. The author has contributed to research in topics: Retinal & Vascular endothelial growth factor. The author has an hindex of 19, co-authored 60 publications receiving 1520 citations. Previous affiliations of Maurizio Cammalleri include Scripps Research Institute.
Papers
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Journal ArticleDOI
Time-restricted role for dendritic activation of the mTOR-p70S6K pathway in the induction of late-phase long-term potentiation in the CA1
Maurizio Cammalleri,Robert Lutjens,Fulvia Berton,Alvin R. King,Cindy Simpson,Walter Francesconi,Pietro Paolo Sanna +6 more
TL;DR: The present data suggest that the N-methyl-d-aspartate-, phosphatidylinositol 3-kinase-dependent dendritic activation of the mTOR-p70S6K pathway is necessary for the induction phase of protein synthesis-dependent synaptic plasticity.
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Phosphatidylinositol 3-Kinase Is Required for the Expression But Not for the Induction or the Maintenance of Long-Term Potentiation in the Hippocampal CA1 Region
Pietro Paolo Sanna,Maurizio Cammalleri,Fulvia Berton,Cindy Simpson,Robert Lutjens,Floyd E. Bloom,Walter Francesconi +6 more
TL;DR: It is observed that PI3-kinase activity was induced in the CA1 region during LTP of field EPSPs (fEPSPs) and that two structurally unrelated PI3 -kinase inhibitors, LY294002 and wortmannin, abated established LTP, suggesting thatPI3-Kinase is involved in the maintenance–expression phase of LTP.
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Role of the adrenergic system in a mouse model of oxygen-induced retinopathy: antiangiogenic effects of beta-adrenoreceptor blockade.
Chiara Ristori,Luca Filippi,Massimo Dal Monte,Davide Martini,Maurizio Cammalleri,Pina Fortunato,Giancarlo la Marca,Patrizio Fiorini,Paola Bagnoli +8 more
TL;DR: This is the first demonstration that β-AR blockade is protective against retinal angiogenesis and ameliorates BRB dysfunction in OIR and may help to establish potential pharmacologic targets based on β3-AR pharmacology.
Journal ArticleDOI
A role for Src kinase in spontaneous epileptiform activity in the CA3 region of the hippocampus
Pietro Paolo Sanna,Fulvia Berton,Maurizio Cammalleri,Melanie K. Tallent,George R. Siggins,Floyd E. Bloom,Walter Francesconi +6 more
TL;DR: A role for Src family PTKs in the pathophysiology of epilepsy is demonstrated and potential therapeutic targets for antiepileptic therapy are suggested.
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The metabotropic glutamate receptor 5 is necessary for late-phase long-term potentiation in the hippocampal CA1 region.
TL;DR: It was found that both LY367385 and MPEP inhibited the induction of an LTP obtained with a single train of tetanization of 1 s duration at 100 Hz, in agreement with previous mouse mutant studies.