M
Mengying Suo
Researcher at Chinese Ministry of Education
Publications - 4
Citations - 55
Mengying Suo is an academic researcher from Chinese Ministry of Education. The author has contributed to research in topics: Medicine & Gene. The author has an hindex of 1, co-authored 2 publications receiving 5 citations.
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Journal ArticleDOI
Dapagliflozin alleviates cardiac fibrosis through suppressing EndMT and fibroblast activation via AMPKα/TGF-β/Smad signalling in type 2 diabetic rats
Jingjing Tian,Mingjun Zhang,Mengying Suo,Dian Liu,Xuyang Wang,Ming Liu,Jinyu Pan,Tao Jin,Fengshuang An +8 more
TL;DR: In this article, the effect of SGLT2 inhibitor dapagliflozin (DAPA) on diabetic cardiomyopathy (DCM) especially for cardiac fibrosis and explore the underlying mechanism.
Journal ArticleDOI
Tongxinluo May Alleviate Inflammation and Improve the Stability of Atherosclerotic Plaques by Changing the Intestinal Flora
Yan Qin,Wenzhao Liu,Xuefang Yan,Chen Zhang,Chunmei Zhang,Lingxin Liu,Xuehui Zheng,Mengying Suo,Yun Ti,Mei Ni,Meng Zhang,Peili Bu +11 more
TL;DR: Tongxinluo can improve plaque stability and reduce inflammation in atherosclerotic rabbits, which may be achieved by modulating intestinal flora and intestinal metabolism.
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SS31 Alleviates Pressure Overload-Induced Heart Failure Caused by Sirt3-Mediated Mitochondrial Fusion
Mengying Suo,Yan Qin,Lingxin Liu,Chunmei Zhang,Jingyuan Li,Xuefang Yan,Chen Zhang,Yun Ti,Tongshuai Chen,Peili Bu +9 more
TL;DR: SS31 can alleviate pressure overload-induced heart failure through Sirt3-mediated mitochondrial fusion, and new directions and drug options for the clinical treatment of heart failure caused by pressure overload are provided.
Journal ArticleDOI
Sirtuin 3 deficiency aggravates angiotensin II-induced hypertensive cardiac injury by the impairment of lymphangiogenesis.
Chen Zhang,Na Li,Mengying Suo,Chunmei Zhang,Jing Liu,Lingxin Liu,Yan Qi,Xuehui Zheng,Lin Xie,Yang Hu,Peili Bu +10 more
TL;DR: In this article, Sirtuin 3 (SIRT3) is an effective mitochondrial deacetylase that has the potential to modulate this process; however, its role in hypertension-induced cardiac lymphangiogenesis to date has not been investigated.