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Michael R. Wessels

Researcher at Heidelberg University

Publications -  764
Citations -  56005

Michael R. Wessels is an academic researcher from Heidelberg University. The author has contributed to research in topics: Large Hadron Collider & Lepton. The author has an hindex of 117, co-authored 761 publications receiving 51429 citations. Previous affiliations of Michael R. Wessels include Beth Israel Deaconess Medical Center & University of Washington.

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Genome analysis of multiple pathogenic isolates of Streptococcus agalactiae: Implications for the microbial “pan-genome”

Hervé Tettelin, +45 more
- 27 Sep 2005 - 
TL;DR: The genomic sequence of six strains representing the five major disease-causing serotypes of Streptococcus agalactiae, the main cause of neonatal infection in humans, was generated and Mathematical extrapolation of the data suggests that the gene reservoir available for inclusion in the S. agalactic pan-genome is vast and that unique genes will continue to be identified even after sequencing hundreds of genomes.
Journal ArticleDOI

Combined Measurement of the Higgs Boson Mass in pp Collisions at √s=7 and 8 TeV with the ATLAS and CMS Experiments

Georges Aad, +5120 more
- 14 May 2015 - 
TL;DR: A measurement of the Higgs boson mass is presented based on the combined data samples of the ATLAS and CMS experiments at the CERN LHC in the H→γγ and H→ZZ→4ℓ decay channels.
Posted Content

Expected Performance of the ATLAS Experiment - Detector, Trigger and Physics

Georges Aad, +2604 more
- 28 Dec 2008 - 
TL;DR: In this article, a detailed study of the expected performance of the ATLAS detector is presented, together with the reconstruction of tracks, leptons, photons, missing energy and jets, along with the performance of b-tagging and the trigger.
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Recognition of pneumolysin by Toll-like receptor 4 confers resistance to pneumococcal infection.

Richard Malley, +9 more
- 18 Feb 2003 - 
TL;DR: The interaction of pneumolysin with TLR4 is critically involved in the innate immune response to pneumococcus and is found to stimulate tumor necrosis factor-α and IL-6 release in wild-type macrophages but not in macrophage from mice with a targeted deletion of the cytoplasmic TLR-adapter molecule myeloid differentiation factor 88, suggesting the involvement of the TLRs in pneumoly sin recognition.
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Complete genome sequence and comparative genomic analysis of an emerging human pathogen, serotype V Streptococcus agalactiae

Hervé Tettelin, +46 more
- 17 Sep 2002 - 
TL;DR: In silico analyses, combined with comparative genome hybridization experiments between the sequenced serotype V strain 2603 V/R and 19 S. agalactiae strains from several serotypes using whole-genome microarrays, revealed the genetic heterogeneity among S. agriculture, provided insights into the evolution of virulence mechanisms.