M
Michael S. Golinko
Researcher at University of Arkansas for Medical Sciences
Publications - 43
Citations - 4898
Michael S. Golinko is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Medicine & Craniosynostosis. The author has an hindex of 16, co-authored 33 publications receiving 4157 citations. Previous affiliations of Michael S. Golinko include Children's Memorial Hospital & Columbia University.
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Journal ArticleDOI
Growth factors and cytokines in wound healing.
TL;DR: A review of the specific roles of these growth factors and cytokines during wound healing can be found in this article, where patients are treated by three growth factors: PDGF-BB, bFGF, and GM-CSF.
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The role of vascular endothelial growth factor in wound healing.
TL;DR: Experimental data supports the hypothesis that VEGF stimulates epithelialization and collagen deposition in a wound, but likely promotes collagen deposition and epithelization as well, and stimulates wound healing through angiogenesis.
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High cost of stage IV pressure ulcers
Harold Brem,Jason Maggi,David M. Nierman,Linda Rolnitzky,David Bell,Robert C. Rennert,Michael S. Golinko,Alan Yan,Courtney Lyder,Bruce C. Vladeck +9 more
TL;DR: The costs incurred from stage IV pressure ulcers are much greater than previously estimated and have the potential to eradicate enormous pain and suffering, save thousands of lives, and reduce health care expenditures by millions of dollars.
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Molecular markers in patients with chronic wounds to guide surgical debridement.
Harold Brem,Olivera Stojadinovic,Robert F. Diegelmann,Hyacinth Entero,Hyacinth Entero,Brian Lee,Irena Pastar,Michael S. Golinko,Harvey J. Rosenberg,Marjana Tomic-Canic +9 more
TL;DR: It is concluded that chronic ulcers contain distinct subpopulations of cells with different capacity to heal and that gene expression profiling can be utilized to identify them, thereby making surgical debridement more accurate and more efficacious.
Journal ArticleDOI
Mechanism of Sustained Release of Vascular Endothelial Growth Factor in Accelerating Experimental Diabetic Healing
Harold Brem,Arber Kodra,Michael S. Golinko,Hyacinth Entero,Olivera Stojadinovic,Vincent M. Wang,Claudia M. Sheahan,Alan D. Weinberg,Savio L. C. Woo,H. Paul Ehrlich,Marjana Tomic-Canic +10 more
TL;DR: ADV-mediated gene transfer accelerates experimental wound healing and VEGF(165), in vitro, stimulates the migration of cultured human keratinocytes and fibroblasts, thus revealing a non-angiogenic effect of V EGF on wound closure.