M
Min Wang
Researcher at Guilin Medical University
Publications - 14
Citations - 442
Min Wang is an academic researcher from Guilin Medical University. The author has contributed to research in topics: Renin–angiotensin system & Diabetes mellitus. The author has an hindex of 7, co-authored 14 publications receiving 228 citations.
Papers
More filters
Journal ArticleDOI
Obesity in patients with COVID-19: a systematic review and meta-analysis.
TL;DR: Obesity increases risk for hospitalization, ICU admission, IMV requirement and death among patients with CO VID-19 and excessive visceral adiposity appears to be associated with severe COVID-19 outcomes.
Journal ArticleDOI
Renin–angiotensin system blockade for the risk of cancer and death
Jian Shen,Yan-Mei Huang,Min Wang,Xue-Zhi Hong,Xin-Nan Song,Xia Zou,Yan-Hong Pan,Wei Ling,Min-Hui Zhu,Xiao-Xi Zhang,Yi Sui,Hai-Lu Zhao +11 more
TL;DR: The significant benefits of renin–angiotensin system blockade observed in case–control studies and cohort studies might diminish in randomised controlled trials.
Journal ArticleDOI
ACE Gene I/D Polymorphism and Obesity in 1,574 Patients with Type 2 Diabetes Mellitus.
Yan-Hong Pan,Min Wang,Yan-Mei Huang,Ying-Hui Wang,Yin-Ling Chen,Li-Jun Geng,Xiao-Xi Zhang,Hai-Lu Zhao +7 more
TL;DR: The relationship of ACE gene I/D polymorphism with obesity is insignificant in Chinese patients with T2DM, and SBP and PP might be higher in the ACE II carriers than in the DD and ID carriers.
Journal ArticleDOI
Wen Dan Decoction for hemorrhagic stroke and ischemic stroke.
TL;DR: TCM such as WDD may serve as a therapeutic tool of dual actions to explore the common mechanisms underlying cerebral hemorrhage and ischemia and displays benefits on improvement of neurological function and overall therapeutic efficacy in post-stroke patients.
Journal ArticleDOI
Renal kallikrein activation and renoprotection after dual blockade of renin-angiotensin system in diet-induced diabetic nephropathy.
TL;DR: Compared with monotherapy, combined treatment further alleviated the kidney impairments in parallel to increased tubular immunoreactivity for kallikrein and decreased immunopositive cells for CD68, TGF-β, and α-SMA.