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Muhammad Salman Shabbir

Researcher at Cork College of Commerce

Publications -  122
Citations -  2106

Muhammad Salman Shabbir is an academic researcher from Cork College of Commerce. The author has contributed to research in topics: Renewable energy & Schiff base. The author has an hindex of 20, co-authored 111 publications receiving 1296 citations. Previous affiliations of Muhammad Salman Shabbir include Universiti Sains Malaysia & Universiti Utara Malaysia.

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Structural Equation Model for Evaluating Factors Affecting Quality of Social Infrastructure Projects

TL;DR: In this article, the authors applied the Structural Equation Modeling (SEM) approach and built a model that explained and identified the critical factors affecting quality in social infrastructure projects.
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Health care service delivery based on the Internet of things: A systematic and comprehensive study

TL;DR: It is found that IoT could help the governments to improve health services in society and commercial interactions and will directly support academics and working professionals for better knowing the progress in IoT mechanisms.
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Exploring the Impact of the COVID-19 Pandemic on University Students’ Learning Life: An Integrated Conceptual Motivational Model for Sustainable and Healthy Online Learning

TL;DR: In this paper, the authors investigated whether the online learning platforms used by university students during the COVID-19 period have presented any challenges to their learning and proposed solutions by developing a conceptual model to reduce the impact of such challenges.
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Predictors for e-government adoption: integrating TAM, TPB, trust and perceived risk

TL;DR: Results indicate that the proposed model is a stable model with powerful explanatory of variation, and some new relationships in the e-government context are found.
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Mutations of human TMHS cause recessively inherited non-syndromic hearing loss

TL;DR: These findings establish the importance of TMHS for normal sound transduction in humans and reveal a homozygous frameshift mutation and a missense mutation in affected individuals of two families segregating non-syndromic deafness, one of which showed significant evidence of linkage to markers in the DFNB67 interval.