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Natalia Castaño-Rodríguez
Researcher at University of New South Wales
Publications - 40
Citations - 2763
Natalia Castaño-Rodríguez is an academic researcher from University of New South Wales. The author has contributed to research in topics: Helicobacter pylori & Microbiome. The author has an hindex of 20, co-authored 32 publications receiving 2056 citations. Previous affiliations of Natalia Castaño-Rodríguez include Del Rosario University.
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Journal ArticleDOI
Global Epidemiology of Campylobacter Infection
TL;DR: Overall, campylobacteriosis is still one of the most important infectious diseases that is likely to challenge global health in the years to come.
Journal ArticleDOI
Faecal Microbiota Transplantation for Inflammatory Bowel Disease: A Systematic Review and Meta-analysis.
Sudarshan Paramsothy,Sudarshan Paramsothy,Ramesh Paramsothy,David T. Rubin,Michael A. Kamm,Nadeem O. Kaakoush,Hazel M. Mitchell,Natalia Castaño-Rodríguez +7 more
TL;DR: FMT appears effective in UC remission induction, but long-term durability and safety remain unclear, and additional well-designed controlled studies of FMT in IBD are needed, especially in CD and pouchitis.
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Dual role of Helicobacter and Campylobacter species in IBD: a systematic review and meta-analysis
TL;DR: Closely related bacteria including EHS and Campylobacter spp increase the risk of IBD, and results infer that H. pylori might exert an immunomodulatory effect in IBD.
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Dysbiosis of the microbiome in gastric carcinogenesis.
Natalia Castaño-Rodríguez,Khean-Lee Goh,Kwong Ming Fock,Hazel M. Mitchell,Nadeem O. Kaakoush +4 more
TL;DR: Three mechanisms of interest in GC are highlighted, including enrichment of pro-inflammatory oral bacterial species, increased abundance of lactic acid producing bacteria, and enrichment of short chain fatty acid production pathways.
Journal ArticleDOI
Pattern-recognition receptors and gastric cancer.
TL;DR: The modulation of PRRs has been suggested to suppress H. pylori-induced inflammation and enhance GC cell apoptosis, highlighting their potential relevance in GC therapeutics.